Morton T H, Natkin E
Department of Oral Biology, University of Washington, School of Dentistry, Seattle.
J Prosthet Dent. 1990 Nov;64(5):539-47. doi: 10.1016/0022-3913(90)90124-u.
A total of 19 hyperostoses underlying mandibular posterior fixed partial dentures were observed in 16 patients. In three patients the lesions were bilateral. Histologic examination of hyperostoses from eight patients showed that they were composed essentially of hyperplastic lamellar bone. Radiographic and clinical evidence suggested that subpontic hyperostoses are chronic, slow growing lesions with an exclusive predilection for the mandibular molar-premolar site. They appear to have variable growth rates and attain widely variable maximum size. The etiology of subpontic hyperostoses is unknown, but it is possible that inflammation, trauma, mandibular and occlusal function, and genetic factors, either individually or in combination, play a role in the initiation and development of these lesions. Subpontic hyperostoses have potential periodontal and restorative implications that may require their surgical removal.
在16例患者中,共观察到19处下颌后牙固定局部义齿下方的骨增生。3例患者的病变为双侧性。对8例患者骨增生的组织学检查显示,它们主要由增生的板层骨组成。影像学和临床证据表明,桥体下骨增生是慢性、生长缓慢的病变,仅好发于下颌磨牙-前磨牙部位。它们似乎有不同的生长速度,最大尺寸差异很大。桥体下骨增生的病因尚不清楚,但炎症、创伤、下颌及咬合功能以及遗传因素单独或共同作用,可能在这些病变的发生和发展中起作用。桥体下骨增生对牙周和修复有潜在影响,可能需要手术切除。