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Different deviation patterns of carbohydrate-metabolizing enzymes in primary rat hepatomas induced by different chemical carcinogens.

作者信息

Sato K, Takaya S, Imai F, Hatayama I, Ito N

出版信息

Cancer Res. 1978 Sep;38(9):3086-93.

PMID:209895
Abstract
摘要

相似文献

1
Different deviation patterns of carbohydrate-metabolizing enzymes in primary rat hepatomas induced by different chemical carcinogens.不同化学致癌物诱导的原发性大鼠肝癌中碳水化合物代谢酶的不同偏差模式。
Cancer Res. 1978 Sep;38(9):3086-93.
2
Early events during liver carcinogenesis involving two carcinogen:protein complexes.肝癌发生过程中涉及两种致癌物：蛋白质复合物的早期事件。
Cancer Res. 1981 Oct;41(10):4039-49.
3
Enzyme deviation patterns in primary rat hepatomas induced by sequential administration of two chemically different carcinogens.连续给予两种化学性质不同的致癌物诱导的原发性大鼠肝癌中的酶偏差模式。
Cancer Res. 1981 Oct;41(10):4147-53.
4
[Binding of 2-acetylaminofluorene-9-14C with rat liver nucleic acids during malignization and in primary hepatomas].[2-乙酰氨基芴-9-¹⁴C在大鼠肝脏恶性转化及原发性肝癌形成过程中与核酸的结合]
Biull Eksp Biol Med. 1977 Mar;83(3):332-4.
5
Altered cobalamin distribution in rat hepatomas and in the livers of rats treated with diethylnitrosamine.大鼠肝癌及经二乙基亚硝胺处理的大鼠肝脏中钴胺素分布的改变
Cancer Res. 1977 Sep;37(9):2975-8.
6
Expression of fibronectin and laminin in the rat liver after partial hepatectomy, during carcinogenesis, and in transplantable hepatocellular carcinomas.部分肝切除术后、致癌过程中以及可移植性肝细胞癌中大鼠肝脏中纤连蛋白和层粘连蛋白的表达。
J Natl Cancer Inst. 1982 Nov;69(5):1105-14.
7
Competitive RNA-DNA hybridization of nuclear and microsomal RNA during diethylnitrosamine hepatocarcinogenesis.二乙基亚硝胺诱导肝癌发生过程中核RNA和微粒体RNA的竞争性RNA-DNA杂交
Cancer Res. 1973 Jul;33(7):1662-9.
8
[Change in antioxidative activity of mice liver lipids during induction of hepatoma with orthoaminoazotoluol].[用邻氨基偶氮甲苯诱发肝癌过程中小鼠肝脏脂质抗氧化活性的变化]
Biofizika. 1968 May-Jun;13(3):443-8.
9
Proceedings: Glucocorticoid receptors and response in diethylnitrosamine-induced hepatomas.
J Endocrinol. 1975 Nov;67(2):30P-31P.
10
[Hyperplastic foci in chemical carcinogenesis].[化学致癌作用中的增生性病灶]
Tsitologiia. 1976 Sep;18(9):1104-10.

引用本文的文献

1
Phenotypic and karyotypic changes induced in cultured rat hepatic epithelial cells that express the "oval" cell phenotype by exposure to N-methyl-N'-nitro-N-nitrosoguanidine.通过暴露于N-甲基-N'-硝基-N-亚硝基胍，在表达“卵圆”细胞表型的培养大鼠肝上皮细胞中诱导的表型和核型变化。
Am J Pathol. 1985 Feb;118(2):306-15.
2
Purification, induction, and distribution of placental glutathione transferase: a new marker enzyme for preneoplastic cells in the rat chemical hepatocarcinogenesis.胎盘谷胱甘肽转移酶的纯化、诱导及分布：大鼠化学性肝癌发生过程中一种新的癌前细胞标志物酶
Proc Natl Acad Sci U S A. 1985 Jun;82(12):3964-8. doi: 10.1073/pnas.82.12.3964.
3
Histochemical and microbiochemical demonstration of reduced pyruvate kinase activity in thioacetamide-induced neoplastic nodules of rat liver.
硫代乙酰胺诱导的大鼠肝脏肿瘤结节中丙酮酸激酶活性降低的组织化学和微生物化学证明
Histochemistry. 1988;90(1):37-42. doi: 10.1007/BF00495704.
4
Glutathione S-transferases and hepatocarcinogenesis.谷胱甘肽S-转移酶与肝癌发生
Jpn J Cancer Res. 1988 May;79(5):556-72. doi: 10.1111/j.1349-7006.1988.tb00022.x.
5
Decrease in glucokinase and glucose-6-phosphatase and increase in hexokinase in putative preneoplastic lesions of rat liver.大鼠肝脏潜在癌前病变中葡萄糖激酶和葡萄糖-6-磷酸酶减少，己糖激酶增加。
J Cancer Res Clin Oncol. 1987;113(5):430-6. doi: 10.1007/BF00390036.