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壳聚糖诱导拟南芥气孔关闭，并伴有过氧化物酶介导的活性氧生成。

Chitosan-induced stomatal closure accompanied by peroxidase-mediated reactive oxygen species production in Arabidopsis.

作者信息

Khokon Md Atiqur Rahman, Uraji Misugi, Munemasa Shintaro, Okuma Eiji, Nakamura Yoshimasa, Mori Izumi C, Murata Yoshiyuki

机构信息

Graduate School of Natural Science and Technology, Division of Bioscience, Okayama University, Japan.

出版信息

Biosci Biotechnol Biochem. 2010;74(11):2313-5. doi: 10.1271/bbb.100340. Epub 2010 Nov 7.

DOI:10.1271/bbb.100340
PMID:21071853
Abstract

Chitosan induced stomatal closure in wild type-plants and NADPH oxidase knock-out mutants (atrbohD atrbohF), and reactive oxygen species (ROS) production in wild-type guard cells. Closure and production were completely abolished by catalase and a peroxidase inhibitor. These results indicate that chitosan induces ROS production mediated by peroxidase, resulting in stomatal closure.

摘要

壳聚糖诱导野生型植物和NADPH氧化酶敲除突变体（atrbohD和atrbohF）的气孔关闭，以及野生型保卫细胞中活性氧（ROS）的产生。过氧化氢酶和过氧化物酶抑制剂完全消除了这种关闭和产生现象。这些结果表明，壳聚糖诱导过氧化物酶介导的ROS产生，从而导致气孔关闭。

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