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2
The mechanism of p21-activated kinase 2 autoactivation.p21激活激酶2的自激活机制。
J Biol Chem. 2003 Oct 24;278(43):41768-78. doi: 10.1074/jbc.M308196200. Epub 2003 Aug 7.
3
Negative regulation of caspase 3-cleaved PAK2 activity by protein phosphatase 1.蛋白磷酸酶1对caspase 3切割的PAK2活性的负调控
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5
Phosphorylation of Mnk1 by caspase-activated Pak2/gamma-PAK inhibits phosphorylation and interaction of eIF4G with Mnk.半胱天冬酶激活的Pak2/γ-PAK对Mnk1的磷酸化作用会抑制eIF4G与Mnk的磷酸化及相互作用。
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PAK2 activated by Cdc42 and caspase 3 mediates different cellular responses to oxidative stress-induced apoptosis.PAK2 被 Cdc42 和 caspase 3 激活,介导细胞对氧化应激诱导的细胞凋亡的不同反应。
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The P21-Activated Kinase 1 and 2 As Potential Therapeutic Targets for the Management of Cardiovascular Disease.p21激活激酶1和2作为心血管疾病治疗的潜在靶点
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5
Myosin 3A kinase activity is regulated by phosphorylation of the kinase domain activation loop.肌球蛋白 3A 激酶活性受激酶结构域激活环磷酸化的调节。
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本文引用的文献

1
PAK signaling in oncogenesis.PAK信号传导在肿瘤发生中的作用
Oncogene. 2009 Jul 16;28(28):2545-55. doi: 10.1038/onc.2009.119. Epub 2009 May 25.
2
Protein kinase activation loop autophosphorylation in cis: overcoming a Catch-22 situation.蛋白激酶激活环顺式自磷酸化:克服两难困境。
Sci Signal. 2009 Jan 20;2(54):pe4. doi: 10.1126/scisignal.254pe4.
3
An isoform-selective, small-molecule inhibitor targets the autoregulatory mechanism of p21-activated kinase.一种亚型选择性小分子抑制剂靶向p21活化激酶的自动调节机制。
Chem Biol. 2008 Apr;15(4):322-31. doi: 10.1016/j.chembiol.2008.03.005.
4
A tale of two Paks.两个巴基斯坦的故事。
Biol Cell. 2008 Feb;100(2):97-108. doi: 10.1042/BC20070109.
5
Activation segment exchange: a common mechanism of kinase autophosphorylation?激活片段交换:激酶自磷酸化的常见机制?
Trends Biochem Sci. 2007 Aug;32(8):351-6. doi: 10.1016/j.tibs.2007.06.004. Epub 2007 Jul 12.
6
A chaperone-dependent GSK3beta transitional intermediate mediates activation-loop autophosphorylation.一种伴侣蛋白依赖性糖原合酶激酶3β过渡中间体介导激活环自磷酸化。
Mol Cell. 2006 Nov 17;24(4):627-33. doi: 10.1016/j.molcel.2006.10.009.
7
p21-activated kinases in cancer.癌症中的p21激活激酶
Nat Rev Cancer. 2006 Jun;6(6):459-71. doi: 10.1038/nrc1892.
8
The Ste5 scaffold allosterically modulates signaling output of the yeast mating pathway.Ste5支架蛋白可别构调节酵母交配途径的信号输出。
Science. 2006 Feb 10;311(5762):822-6. doi: 10.1126/science.1120941. Epub 2006 Jan 19.
9
Activation-loop autophosphorylation is mediated by a novel transitional intermediate form of DYRKs.激活环自身磷酸化由一种新型的双重特异性酪氨酸磷酸化调节激酶(DYRKs)过渡中间形式介导。
Cell. 2005 Jun 17;121(6):925-36. doi: 10.1016/j.cell.2005.03.034.
10
Regulation of protein kinases; controlling activity through activation segment conformation.蛋白激酶的调控;通过激活片段构象控制活性。
Mol Cell. 2004 Sep 10;15(5):661-75. doi: 10.1016/j.molcel.2004.08.024.

PAK2 自身激活的机制研究:顺式起始随后反式扩增的两步模型。

Mechanistic studies of the autoactivation of PAK2: a two-step model of cis initiation followed by trans amplification.

机构信息

MOE Key Laboratory for Bioinformatics, School of Life Sciences, Tsinghua University, Beijing 100084, PR China.

出版信息

J Biol Chem. 2011 Jan 28;286(4):2689-95. doi: 10.1074/jbc.M110.156505. Epub 2010 Nov 22.

DOI:10.1074/jbc.M110.156505
PMID:21098037
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3024765/
Abstract

Protein kinase activation, via autophosphorylation of the activation loop, is a common regulatory mechanism in phosphorylation-dependent signaling cascades. Despite the prevalence of this reaction and its importance in biological regulation, the molecular mechanisms of autophosphorylation are poorly understood. In this study, we developed a kinetic approach to distinguish quantitatively between cis- and trans-pathways in an autocatalytic reaction. Using this method, we have undertaken a detailed kinetic analysis for the autoactivation mechanism of p21-activated protein kinase 2 (PAK2). PAK2 is regulated in vivo and in vitro by small GTP-binding proteins, Cdc42 and Rac. Full activation of PAK2 requires autophosphorylation of the conserved threonine, Thr(402), in the activation loop of its catalytic kinase domain. Analyses of the time courses of substrate reaction during PAK2 autoactivation suggest that autophosphorylation of Thr(402) in PAK2 obeys a two-step mechanism of cis initiation, followed by trans amplification. The unphosphorylated PAK2 undergoes an intramolecular (cis) autophosphorylation on Thr(402) to produce phosphorylated PAK2, and this newly formed active PAK2 then phosphorylates other PAK2 molecules at Thr(402) in an intermolecular (trans) manner. Based on the kinetic equation derived, all microscopic kinetic constants for the cis and trans autophosphorylation have been estimated quantitatively. The advantage of the new method is not only its usefulness in the study of fast activation reactions, but its convenience in the study of substrate effects on modification reaction. It would be particularly useful when the regulatory mechanism of the autophosphorylation reaction toward certain enzymes is being assessed.

摘要

蛋白激酶的激活是磷酸化依赖的信号级联反应中的一种常见调节机制,通过激活环的自身磷酸化来实现。尽管这种反应很普遍,而且在生物调节中非常重要,但自身磷酸化的分子机制仍未被充分理解。在这项研究中,我们开发了一种动力学方法,可以定量区分自催化反应中的顺式和反式途径。我们使用这种方法,对 p21 激活蛋白激酶 2(PAK2)的自动激活机制进行了详细的动力学分析。PAK2 在体内和体外受到小分子 GTP 结合蛋白 Cdc42 和 Rac 的调节。PAK2 的完全激活需要其催化激酶结构域中激活环上的保守苏氨酸 Thr(402)的自身磷酸化。对 PAK2 自动激活过程中底物反应的时间进程进行分析表明,PAK2 中 Thr(402)的自身磷酸化遵循顺式起始、反式放大的两步机制。未磷酸化的 PAK2 在 Thr(402)上发生分子内(顺式)自身磷酸化,产生磷酸化的 PAK2,然后这种新形成的活性 PAK2 以分子间(反式)方式磷酸化其他 PAK2 分子上的 Thr(402)。根据推导的动力学方程,定量估计了顺式和反式自身磷酸化的所有微观动力学常数。新方法的优点不仅在于它在快速激活反应研究中的有用性,还在于它在修饰反应中研究底物效应的便利性。当评估某些酶的自身磷酸化反应的调节机制时,它将特别有用。