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G protein mediated regulation of K+ channels in heart.

作者信息

Szabo G, Otero A S

机构信息

Department of Physiology and Biophysics, University of Texas Medical Branch, Galveston 77550-2781.

出版信息

Annu Rev Physiol. 1990;52:293-305. doi: 10.1146/annurev.ph.52.030190.001453.

DOI:10.1146/annurev.ph.52.030190.001453
PMID:2109964
Abstract
摘要

相似文献

1
G protein mediated regulation of K+ channels in heart.G蛋白介导的心脏钾通道调节
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2
Ionic channels and their regulation by G protein subunits.离子通道及其受G蛋白亚基的调节。
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A functional model for G protein activation of the muscarinic K+ channel in guinea pig atrial myocytes. Spectral analysis of the effect of GTP on single-channel kinetics.豚鼠心房肌细胞中毒蕈碱型钾通道G蛋白激活的功能模型。GTP对单通道动力学影响的频谱分析。
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G protein regulation of cardiac muscarinic potassium channel.G蛋白对心肌毒蕈碱钾通道的调节作用。
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[Regulation of muscarinic potassium channels].[毒蕈碱钾通道的调节]
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G protein regulation of potassium ion channels.G蛋白对钾离子通道的调节。
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Inactivation of a G protein-coupled inwardly rectifying K+ channel.一种G蛋白偶联内向整流钾通道的失活
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[On the regulation of cardiac K channel by GTP-binding proteins--basic mechanism and clinical aspect of acetylcholine and adenosine-increase of cardiac potassium conductance].[关于GTP结合蛋白对心脏钾通道的调节——乙酰胆碱和腺苷增加心脏钾电导的基本机制及临床意义]
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Muscarinic activation of potassium channels in cardiac myocytes: kinetic aspects of G protein function in vivo.心肌细胞中钾通道的毒蕈碱激活：体内G蛋白功能的动力学方面
Trends Pharmacol Sci. 1989 Dec;Suppl:46-9.

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Serum contains a potent factor that decreases beta-adrenergic receptor-stimulated L-type Ca2+ current in cardiac myocytes.血清中含有一种强效因子，可降低心肌细胞中β-肾上腺素能受体刺激的L型钙电流。
Pflugers Arch. 1993 May;423(3-4):245-50. doi: 10.1007/BF00374402.
2
Agonist-independent effects of muscarinic antagonists on Ca2+ and K+ currents in frog and rat cardiac cells.毒蕈碱拮抗剂对青蛙和大鼠心肌细胞钙电流和钾电流的非激动剂依赖性作用。
J Physiol. 1993 Feb;461:743-65. doi: 10.1113/jphysiol.1993.sp019539.
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Activation of muscarinic K+ current in guinea-pig atrial myocytes by a serum factor.
血清因子对豚鼠心房肌细胞毒蕈碱钾电流的激活作用
J Physiol. 1993 Feb;461:263-81. doi: 10.1113/jphysiol.1993.sp019513.
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On the role of G protein activation and phosphorylation in desensitization to acetylcholine in guinea-pig atrial cells.关于G蛋白激活和磷酸化在豚鼠心房细胞对乙酰胆碱脱敏中的作用
J Physiol. 1993 May;464:649-79. doi: 10.1113/jphysiol.1993.sp019656.
5
Differential time course for desensitization to muscarinic effects on K+ and Ca2+ channels.对毒蕈碱对钾离子和钙离子通道作用脱敏的不同时间进程。
Pflugers Arch. 1994 Oct;428(5-6):542-51. doi: 10.1007/BF00374576.
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A comparative analysis of the time course of cardiac Ca2+ current response to rapid applications of beta-adrenergic and dihydropyridine agonists.对β-肾上腺素能激动剂和二氢吡啶激动剂快速应用时心脏Ca2+电流反应时间进程的比较分析。
Naunyn Schmiedebergs Arch Pharmacol. 1993 Aug;348(2):197-206. doi: 10.1007/BF00164799.
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Early afterdepolarizations in cardiac myocytes: mechanism and rate dependence.心肌细胞早期后去极化：机制与频率依赖性
Biophys J. 1995 Mar;68(3):949-64. doi: 10.1016/S0006-3495(95)80271-7.
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Barium block of the muscarinic potassium current in guinea-pig atrial cells.豚鼠心房细胞中蕈毒碱钾电流的钡阻滞
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J Physiol. 1991 Feb;433:589-613. doi: 10.1113/jphysiol.1991.sp018445.
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Single cardiac outwardly rectifying K+ channels modulated by protein kinase A and a G-protein.由蛋白激酶A和一种G蛋白调节的单个心脏外向整流钾通道
Eur Biophys J. 1991;20(5):281-6. doi: 10.1007/BF00450563.