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Endoplasmic reticulum stress and the unfolded protein response in nonalcoholic fatty liver disease.内质网应激与非酒精性脂肪性肝病中的未折叠蛋白反应。
Antioxid Redox Signal. 2011 Jul 15;15(2):505-21. doi: 10.1089/ars.2010.3790. Epub 2011 Apr 26.
2
Endoplasmic reticulum stress in nonalcoholic fatty liver disease.内质网应激与非酒精性脂肪性肝病。
Annu Rev Nutr. 2012 Aug 21;32:17-33. doi: 10.1146/annurev-nutr-071811-150644.
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Role of endoplasmic reticulum stress in the pathogenesis of nonalcoholic fatty liver disease.内质网应激在非酒精性脂肪性肝病发病机制中的作用
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The adaptive endoplasmic reticulum stress response to lipotoxicity in progressive human nonalcoholic fatty liver disease.渐进性人类非酒精性脂肪性肝病中脂肪毒性的适应性内质网应激反应。
Toxicol Sci. 2014 Jan;137(1):26-35. doi: 10.1093/toxsci/kft230. Epub 2013 Oct 4.
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Endoplasmic reticulum stress and Oxidative stress in the pathogenesis of Non-alcoholic fatty liver disease.内质网应激与氧化应激在非酒精性脂肪性肝病发病机制中的作用
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Fatty acids and the endoplasmic reticulum in nonalcoholic fatty liver disease.脂肪酸与内质网在非酒精性脂肪肝病中的作用。
Biofactors. 2011 Jan-Feb;37(1):8-16. doi: 10.1002/biof.135. Epub 2010 Dec 2.
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The unfolded protein response in fatty liver disease.未折叠蛋白反应与脂肪肝疾病。
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Endoplasmic reticulum proteostasis in hepatic steatosis.内质网蛋白稳态与肝脂肪变性。
Nat Rev Endocrinol. 2016 Dec;12(12):710-722. doi: 10.1038/nrendo.2016.124. Epub 2016 Aug 12.
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Enhanced endoplasmic reticulum SERCA activity by overexpression of hepatic stimulator substance gene prevents hepatic cells from ER stress-induced apoptosis.过表达肝刺激物质基因增强内质网 SERCA 活性可防止肝细胞发生内质网应激诱导的细胞凋亡。
Am J Physiol Cell Physiol. 2014 Feb 1;306(3):C279-90. doi: 10.1152/ajpcell.00117.2013. Epub 2013 Nov 27.
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Endoplasmic Reticulum Stress Related Molecular Mechanisms in Nonalcoholic Fatty Liver Disease (NAFLD).内质网应激相关分子机制在非酒精性脂肪性肝病（NAFLD）中的作用。
Curr Drug Targets. 2018;19(9):1087-1094. doi: 10.2174/1389450118666180516122517.
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Effects of Grape By-Products on Oxidative Stress and Inflammation in Farm Animals: An Overview of Studies Performed in Pigs, Chickens, and Cattle.葡萄副产品对农场动物氧化应激和炎症的影响：猪、鸡和牛的研究综述
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Hepatocellular carcinoma: pathogenesis, molecular mechanisms, and treatment advances.肝细胞癌：发病机制、分子机制及治疗进展
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Therapeutic Targets and Approaches to Manage Inflammation of NAFLD.非酒精性脂肪性肝病炎症管理的治疗靶点与方法
Biomedicines. 2025 Feb 6;13(2):393. doi: 10.3390/biomedicines13020393.
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polysaccharides alleviate metabolic dysfunction-associated steatotic liver disease through enhancing hepatocyte RelA/ HNF1α signaling.多糖通过增强肝细胞RelA/HNF1α信号传导来缓解代谢功能障碍相关的脂肪性肝病。
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A Comprehensive Review on Non-Alcoholic Fatty Liver Disease.非酒精性脂肪性肝病综合综述
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Mitochondrial Cholesterol Metabolites in a Bile Acid Synthetic Pathway Drive Nonalcoholic Fatty Liver Disease: A Revised "Two-Hit" Hypothesis.胆汁酸合成途径中的线粒体胆固醇代谢物驱动非酒精性脂肪性肝病：修正的“双重打击”假说。
Cells. 2023 May 20;12(10):1434. doi: 10.3390/cells12101434.
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DHA induces adipocyte lipolysis through endoplasmic reticulum stress and the cAMP/PKA signaling pathway in grass carp ().二十二碳六烯酸通过内质网应激和草鱼中的环磷酸腺苷/蛋白激酶A信号通路诱导脂肪细胞脂解（）。
Anim Nutr. 2022 Dec 31;13:185-196. doi: 10.1016/j.aninu.2022.10.010. eCollection 2023 Jun.
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Fatty Acid Excess Dysregulates CARF to Initiate the Development of Hepatic Steatosis.脂肪酸过量会扰乱 CARF 从而引发肝脂肪变性。
Cells. 2023 Apr 1;12(7):1069. doi: 10.3390/cells12071069.
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Non-Alcoholic Fatty Liver Disease (NAFLD) Pathogenesis and Natural Products for Prevention and Treatment.非酒精性脂肪性肝病（NAFLD）发病机制及天然产物的防治。
Int J Mol Sci. 2022 Dec 7;23(24):15489. doi: 10.3390/ijms232415489.
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GRP78 Activity Moderation as a Therapeutic Treatment against Obesity.GRP78 活性调节作为肥胖症的治疗方法。
Int J Environ Res Public Health. 2022 Nov 30;19(23):15965. doi: 10.3390/ijerph192315965.
本文引用的文献
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Hepatic gene networks in morbidly obese patients with nonalcoholic fatty liver disease.非酒精性脂肪性肝病肥胖患者的肝脏基因网络。
Obes Surg. 2010 Dec;20(12):1698-709. doi: 10.1007/s11695-010-0171-6.
2
Flavonol activation defines an unanticipated ligand-binding site in the kinase-RNase domain of IRE1.黄酮醇的激活作用定义了 IRE1 激酶-RNase 结构域中一个意想不到的配体结合位点。
Mol Cell. 2010 Apr 23;38(2):291-304. doi: 10.1016/j.molcel.2010.04.001.
3
Distinct gene expression profiles characterize cellular responses to palmitate and oleate.不同的基因表达谱特征描绘了细胞对棕榈酸和油酸的反应。
J Lipid Res. 2010 Aug;51(8):2121-31. doi: 10.1194/jlr.M004275. Epub 2010 Apr 20.
4
Insulin resistance in nonalcoholic fatty liver disease.非酒精性脂肪性肝病中的胰岛素抵抗。
Curr Pharm Des. 2010 Jun;16(17):1941-51. doi: 10.2174/138161210791208875.
5
The regulatory subunits of PI3K, p85alpha and p85beta, interact with XBP-1 and increase its nuclear translocation.PI3K 的调节亚基 p85alpha 和 p85beta 与 XBP-1 相互作用，并增加其核转位。
Nat Med. 2010 Apr;16(4):429-37. doi: 10.1038/nm.2099. Epub 2010 Mar 28.
6
A regulatory subunit of phosphoinositide 3-kinase increases the nuclear accumulation of X-box-binding protein-1 to modulate the unfolded protein response.磷酸肌醇 3-激酶的调节亚基增加 X 盒结合蛋白-1 的核积累，从而调节未折叠蛋白反应。
Nat Med. 2010 Apr;16(4):438-45. doi: 10.1038/nm.2121. Epub 2010 Mar 28.
7
Endoplasmic reticulum stress and the inflammatory basis of metabolic disease.内质网应激与代谢性疾病的炎症基础。
Cell. 2010 Mar 19;140(6):900-17. doi: 10.1016/j.cell.2010.02.034.
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Rapamycin inhibits postprandial-mediated X-box-binding protein-1 splicing in rat liver.雷帕霉素抑制大鼠肝餐后介导的 X 盒结合蛋白-1 剪接。
J Nutr. 2010 May;140(5):879-84. doi: 10.3945/jn.109.119883. Epub 2010 Mar 17.
9
Linking endoplasmic reticulum stress to cell death in hepatocytes: roles of C/EBP homologous protein and chemical chaperones in palmitate-mediated cell death.将内质网应激与肝细胞死亡联系起来：C/EBP 同源蛋白和化学伴侣在软脂酸介导的细胞死亡中的作用。
Am J Physiol Endocrinol Metab. 2010 May;298(5):E1027-35. doi: 10.1152/ajpendo.00642.2009. Epub 2010 Feb 16.
10
Double-stranded RNA-dependent protein kinase links pathogen sensing with stress and metabolic homeostasis.双链 RNA 依赖性蛋白激酶将病原体感应与应激和代谢稳态联系起来。
Cell. 2010 Feb 5;140(3):338-48. doi: 10.1016/j.cell.2010.01.001.
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