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Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system. Activated intrarenal renin-angiotensin system is correlated with high blood pressure in humans.关于“针锋相对”观点的评论：系统性高血压的主要促成因素：交感神经系统的慢性激活与肾内肾素-血管紧张素系统的激活。激活的肾内肾素-血管紧张素系统与人类高血压相关。
J Appl Physiol (1985). 2010 Dec;109(6):2003. doi: 10.1152/japplphysiol.01160.2010.
2
Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system. Activation of the sympathetic nervous system is the dominant contributor to systemic hypertension.关于“针锋相对”的评论：系统性高血压的主要促成因素：交感神经系统的慢性激活与肾内肾素 - 血管紧张素系统的激活。交感神经系统的激活是系统性高血压的主要促成因素。
J Appl Physiol (1985). 2010 Dec;109(6):2006.
3
Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system.关于“针锋相对：系统性高血压的主要促成因素：交感神经系统的慢性激活与肾内肾素 - 血管紧张素系统的激活”的评论
J Appl Physiol (1985). 2010 Dec;109(6):2006-7. doi: 10.1152/japplphysiol.01160.2010.
4
Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system. Sympathetic nervous system, plasma volume, and hypertension.关于“针锋相对：系统性高血压的主要促成因素：交感神经系统的慢性激活与肾内肾素 - 血管紧张素系统的激活”的评论。交感神经系统、血容量与高血压。
J Appl Physiol (1985). 2010 Dec;109(6):2003. doi: 10.1152/japplphysiol.01160.2010.
5
Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system. Dominant contributors for systemic hypertension--who leads, who follows?
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Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system.关于“正方观点：反方观点：系统性高血压的主要促成因素：交感神经系统的慢性激活与肾内肾素 - 血管紧张素系统的激活”的评论
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Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system. Systemic hypertension--a "ratologist" point of view.
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Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system. Chronic activation of sympathetic nervous system and systemic hypertension--stress shows the way.关于“针锋相对：系统性高血压的主要促成因素：交感神经系统的慢性激活与肾内肾素 - 血管紧张素系统的激活”的评论。交感神经系统的慢性激活与系统性高血压——应激指明方向。
J Appl Physiol (1985). 2010 Dec;109(6):2004-5. doi: 10.1152/japplphysiol.01160.2010.
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Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system. Intrarenal angiotensin II generation as a hypertensinogenic mechanism.关于“针锋相对：系统性高血压的主要促成因素：交感神经系统的慢性激活与肾内肾素-血管紧张素系统的激活”的评论。肾内血管紧张素II的生成作为一种致高血压机制。
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Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system. Systemic hypertension--involvement of the CNS to this complex puzzle.关于“针锋相对：系统性高血压的主要促成因素：交感神经系统的慢性激活与肾内肾素 - 血管紧张素系统的激活”的评论。系统性高血压——中枢神经系统在这个复杂难题中的作用。
J Appl Physiol (1985). 2010 Dec;109(6):2004. doi: 10.1152/japplphysiol.01160.2010.

本文引用的文献

1
Counterpoint: Activation of the intrarenal renin-angiotensin system is the dominant contributor to systemic hypertension.反驳观点：肾内肾素-血管紧张素系统的激活是系统性高血压的主要促成因素。
J Appl Physiol (1985). 2010 Dec;109(6):1998-2000; discussion 2015. doi: 10.1152/japplphysiol.00182.2010a.
2
Intrarenal angiotensin-converting enzyme induces hypertension in response to angiotensin I infusion.肾内血管紧张素转换酶在血管紧张素 I 输注时引起高血压。
J Am Soc Nephrol. 2011 Mar;22(3):449-59. doi: 10.1681/ASN.2010060624. Epub 2010 Nov 29.
3
Pressor responses to antihypertensive drug types.降压药类型引起的升压反应。
Am J Hypertens. 2010 Sep;23(9):1031-7. doi: 10.1038/ajh.2010.114. Epub 2010 Aug 19.
4
An arcuate-ventrolateral periaqueductal gray reciprocal circuit participates in electroacupuncture cardiovascular inhibition.弧形腹外侧导水管周围灰质的交互回路参与电针心血管抑制。
Auton Neurosci. 2010 Dec 8;158(1-2):13-23. doi: 10.1016/j.autneu.2010.05.006. Epub 2010 Jun 30.
5
Sustained suppression of sympathetic activity and arterial pressure during chronic activation of the carotid baroreflex.慢性激活颈动脉压力感受器时交感活性和动脉压的持续抑制。
Am J Physiol Heart Circ Physiol. 2010 Aug;299(2):H402-9. doi: 10.1152/ajpheart.00372.2010. Epub 2010 May 28.
6
Neural mechanism of electroacupuncture's hypotensive effects.电针降压作用的神经机制。
Auton Neurosci. 2010 Oct 28;157(1-2):24-30. doi: 10.1016/j.autneu.2010.03.015. Epub 2010 May 5.
7
Mechanisms of obesity-induced hypertension.肥胖导致高血压的机制。
Hypertens Res. 2010 May;33(5):386-93. doi: 10.1038/hr.2010.9.
8
Is it the blood pressure or the blood vessel?是血压还是血管的问题？
J Am Soc Hypertens. 2007 Jan-Feb;1(1):5-16. doi: 10.1016/j.jash.2006.11.001.
9
Neurogenic hypertension.
Exp Physiol. 2010 May;95(5):569-71. doi: 10.1113/expphysiol.2009.047282.
10
Electroacupuncture enhances preproenkephalin mRNA expression in rostral ventrolateral medulla of rats.电针对大鼠延髓头端腹外侧区前强啡肽原 mRNA 表达的增强作用。
Neurosci Lett. 2010 Jun 21;477(2):61-5. doi: 10.1016/j.neulet.2010.04.025. Epub 2010 Apr 24.

Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system. Activated intrarenal renin-angiotensin system is correlated with high blood pressure in humans.

作者信息

Kobori Hiroyuki, Fu Qi, Crowley Steven D, Gonzalez-Villalobos Romer A, Campos Ruy R

机构信息

Department of Medicine, Tulane University Health Sciences Center, LA, USA.

出版信息

J Appl Physiol (1985). 2010 Dec;109(6):2003. doi: 10.1152/japplphysiol.01160.2010.

DOI:10.1152/japplphysiol.01160.2010
PMID:21148352
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3774210/
Abstract
摘要