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人源产超广谱β-内酰胺酶大肠杆菌克隆中复合 IS26-sul3 元件与高可传播 IncI1 质粒的关联。

Association of composite IS26-sul3 elements with highly transmissible IncI1 plasmids in extended-spectrum-beta-lactamase-producing Escherichia coli clones from humans.

机构信息

Servicio de Microbiología, IRYCIS, Madrid, Spain.

出版信息

Antimicrob Agents Chemother. 2011 May;55(5):2451-7. doi: 10.1128/AAC.01448-10. Epub 2011 Feb 22.

DOI:10.1128/AAC.01448-10
PMID:21343460
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3088239/
Abstract

The association of an IS440-sul3 platform with Tn21 class 1 integrons carried by IncI1 plasmids encoding extended-spectrum β-lactamases (ESBLs; mainly SHV-12 and CTX-M-14) among worldwide Escherichia coli clones of phylogroups A (ST10, ST23, and ST46), B1 (ST155, ST351, and ST359), and D/B2 (ST131) is reported. An in silico comparative analysis of sul3 elements available in the GenBank database shows the evolution of sul3 platforms by hosting different transposable elements facilitating the potential genesis of IS26 composite transposons and further insertion element-mediated promoted arrangements.

摘要

报告了在肠杆菌科 A 群（ST10、ST23 和 ST46）、B1 群（ST155、ST351 和 ST359）和 D/B2 群（ST131）的携带编码超广谱β-内酰胺酶（主要为 SHV-12 和 CTX-M-14）的 IncI1 质粒上的 Tn21 类 1 整合子的 IS440-sul3 平台与全球大肠杆菌克隆之间的关联。对 GenBank 数据库中可用的 sul3 元件的计算机比较分析表明，sul3 平台通过容纳不同的转座元件进行进化，从而促进了 IS26 复合转座子的潜在发生，并进一步促进了插入元件介导的排列。

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Multiresistant Gram-negative bacteria: the role of high-risk clones in the dissemination of antibiotic resistance.多重耐药革兰氏阴性菌：高危克隆在抗生素耐药性传播中的作用。
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Prevalence and characterization of plasmids carrying sulfonamide resistance genes among Escherichia coli from pigs, pig carcasses and human.猪、猪胴体和人源大肠杆菌中携带磺胺类药物抗性基因的质粒的流行情况及特征
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Isolation and characterization of potentially pathogenic antimicrobial-resistant Escherichia coli strains from chicken and pig farms in Spain.从西班牙的鸡场和猪场中分离和鉴定具有潜在致病性和抗微生物耐药性的大肠杆菌菌株。
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Class 1 integrons lacking qacEDelta1 and sul1 genes in Escherichia coli isolates of food, animal and human origins.食品、动物和人类来源的大肠杆菌分离株中缺乏 qacEDelta1 和 sul1 基因的 1 类整合子。
Vet Microbiol. 2010 Aug 26;144(3-4):493-7. doi: 10.1016/j.vetmic.2010.01.026. Epub 2010 Feb 4.
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Little evidence for reversibility of trimethoprim resistance after a drastic reduction in trimethoprim use.在大幅减少甲氧苄啶使用后，甲氧苄啶耐药性的可逆性证据很少。
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