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遗传性线粒体神经病变。

Inherited mitochondrial neuropathies.

机构信息

Krankenanstalt Rudolfstiftung, Vienna, Austria.

出版信息

J Neurol Sci. 2011 May 15;304(1-2):9-16. doi: 10.1016/j.jns.2011.02.012. Epub 2011 Mar 13.

DOI:10.1016/j.jns.2011.02.012
PMID:21402391
Abstract

Mitochondrial disorders (MIDs) occasionally manifest as polyneuropathy either as the dominant feature or as one of many other manifestations (inherited mitochondrial neuropathy). MIDs in which polyneuropathy is the dominant feature, include NARP syndrome due to the transition m.8993T>, CMT2A due to MFN2 mutations, CMT2K and CMT4A due to GDAP1 mutations, and axonal/demyelinating neuropathy with external ophthalmoplegia due to POLG1 mutations. MIDs in which polyneuropathy is an inconstant feature among others is the MELAS syndrome, MERRF syndrome, LHON, Mendelian PEO, KSS, Leigh syndrome, MNGIE, SANDO; MIRAS, MEMSA, AHS, MDS (hepato-cerebral form), IOSCA, and ADOA syndrome. In the majority of the cases polyneuropathy presents in a multiplex neuropathy distribution. Nerve conduction studies may reveal either axonal or demyelinated or mixed types of neuropathies. If a hereditary neuropathy is due to mitochondrial dysfunction, the management of these patients is at variance from non-mitochondrial hereditary neuropathies. Patients with mitochondrial hereditary neuropathy need to be carefully investigated for clinical or subclinical involvement of other organs or systems. Supportive treatment with co-factors, antioxidants, alternative energy sources, or lactate lowering agents can be tried. Involvement of other organs may require specific treatment. Mitochondrial neuropathies should be included in the differential diagnosis of hereditary neuropathies.

摘要

线粒体疾病(MIDs)偶尔表现为多发性神经病,要么是主要特征,要么是许多其他表现之一(遗传性线粒体神经病)。以多发性神经病为主要特征的 MIDs 包括由于 m.8993T> 过渡引起的 NARP 综合征、由于 MFN2 突变引起的 CMT2A、由于 GDAP1 突变引起的 CMT2K 和 CMT4A,以及由于 POLG1 突变引起的轴索性/脱髓鞘神经病伴外眼肌麻痹。以多发性神经病为其他表现之一的不恒定特征的 MIDs 包括 MELAS 综合征、MERRF 综合征、LHON、孟德尔 PEO、KSS、 Leigh 综合征、MNGIE、SANDO;MIRAS、MEMSA、AHS、MDS(肝脑型)、IOSCA 和 ADOA 综合征。在大多数情况下,多发性神经病表现为多灶性神经病分布。神经传导研究可能显示轴索性、脱髓鞘性或混合性神经病。如果遗传性神经病是由于线粒体功能障碍引起的,那么这些患者的管理与非线粒体遗传性神经病不同。线粒体遗传性神经病患者需要仔细检查其他器官或系统是否有临床或亚临床受累。可以尝试使用辅助因子、抗氧化剂、替代能源或降低乳酸盐的药物进行支持性治疗。其他器官的受累可能需要特定的治疗。线粒体神经病应包括在遗传性神经病的鉴别诊断中。

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