类风湿关节炎中类风湿因子对Fcγ受体（CD16）阳性自然杀伤细胞的激活作用
[Activation of Fc gamma receptor (CD16) positive natural killer cells by rheumatoid factors in rheumatoid arthritis].
作者信息
Kuipers J G, Hendrich C, Kolanus W, Hammer M, Schmidt R E
机构信息
Medizinische Hochschule Hannover, Abteilung Immunologie und Transfusionsmedizin.
出版信息
Immun Infekt. 1990 Apr;18(2):50-2.
PMID:2141002
Abstract
Fc gamma-receptor-(CD16-)positive natural killer (NK) cells are activated by rheumatoid factor in the synovial fluid of patients with rheumatoid arthritis. This activation induces modulation of the CD16 molecule and release of IFN gamma and TNF alpha by NK cells.
摘要
Fcγ受体（CD16）阳性的自然杀伤（NK）细胞被类风湿性关节炎患者滑液中的类风湿因子激活。这种激活诱导NK细胞的CD16分子发生调节变化，并释放γ干扰素和肿瘤坏死因子α。
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