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新型 CCR5 拮抗剂 TBR-652 在 HIV-1 感染、抗逆转录病毒治疗经验丰富、CCR5 拮抗剂初治患者中的药代动力学和药效学。

Pharmacokinetics and pharmacodynamics of TBR-652, a novel CCR5 antagonist, in HIV-1-infected, antiretroviral treatment-experienced, CCR5 antagonist-naïve patients.

机构信息

Pharsight, a Certara Company, Montreal, Quebec, Canada.

出版信息

Antimicrob Agents Chemother. 2011 Jun;55(6):2768-74. doi: 10.1128/AAC.00713-10. Epub 2011 Apr 12.

Abstract

TBR-652 is a novel CCR5 antagonist with potent in vitro anti-HIV activity. The objective of this study was to determine the pharmacokinetics (PK) and pharmacodynamics (PD) of TBR-652 in HIV-1-infected, antiretroviral treatment-experienced, CCR5 antagonist-naïve patients. A double-blind, placebo-controlled, randomized, dose-escalating study of TBR-652 monotherapy given once daily orally for 10 days was performed, followed by a 40-day follow-up period. Approximately 10 patients/dose level received 25, 50, 75, 100, and 150 mg TBR-652 or placebo (4:1). Blood was collected at different intervals for PK and HIV-1 RNA assessments. PK analysis of TBR-652 was performed using noncompartmental methods. PK/PD was modeled using a maximum inhibitory effect model (E(max)) and 50% inhibitory concentrations (IC₅₀). TBR-652 was well absorbed in the systemic circulation. TBR-652 concentration levels declined slowly, with mean elimination half-lives ranging from 22.5 to 47.62 h across dose levels. TBR-652 treatment resulted in potent, dose-dependent decreases in viral load, with statistically significant decreases in nadir HIV-1 RNA compared to baseline for all dose levels. Suppression of HIV-1 RNA persisted over the 40-day follow-up period. A steep exposure-effect relationship was observed, with an E(max) of -1.43 log₁₀ copies/ml and IC₅₀ of 13.1 ng/ml. TBR-652 was generally safe and well tolerated at all dose levels studied. Short-term monotherapy treatments of TBR-652 in HIV-1-infected patients resulted in promising PK and PD results, with a clear exposure-response relationship at the current dose levels studied. Data from this study support further development of TBR-652 in HIV-infected patients.

摘要

TBR-652 是一种新型的 CCR5 拮抗剂,具有强大的体外抗 HIV 活性。本研究的目的是确定 TBR-652 在 HIV-1 感染、抗逆转录病毒治疗经验丰富、CCR5 拮抗剂初治的患者中的药代动力学(PK)和药效动力学(PD)。进行了一项 TBR-652 单药治疗的双盲、安慰剂对照、随机、剂量递增的研究,患者每天口服 10 天,随后进行 40 天的随访期。大约 10 名患者/剂量水平接受 25、50、75、100 和 150 mg TBR-652 或安慰剂(4:1)。在不同时间点采集血液进行 PK 和 HIV-1 RNA 评估。采用非房室分析方法对 TBR-652 的 PK 进行分析。采用最大抑制效应模型(E(max))和 50%抑制浓度(IC₅₀)对 PK/PD 进行建模。TBR-652 在全身循环中被很好地吸收。TBR-652 浓度水平下降缓慢,各剂量水平的平均消除半衰期范围为 22.5 至 47.62 小时。TBR-652 治疗导致病毒载量呈剂量依赖性下降,与基线相比,所有剂量水平的 HIV-1 RNA 最低点均有统计学意义下降。在 40 天的随访期间,HIV-1 RNA 的抑制持续存在。观察到陡峭的暴露-效应关系,E(max)为-1.43 log₁₀ 拷贝/ml,IC₅₀为 13.1 ng/ml。在研究的所有剂量水平下,TBR-652 通常安全且耐受良好。在 HIV-1 感染患者中,TBR-652 的短期单药治疗产生了有希望的 PK 和 PD 结果,在目前研究的剂量水平下,存在明确的暴露-反应关系。这项研究的数据支持在 HIV 感染患者中进一步开发 TBR-652。

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