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Physical exercise in amyotrophic lateral sclerosis: a potential co-adjuvant therapeutic option to counteract disease progression.肌萎缩侧索硬化症中的体育锻炼：一种对抗疾病进展的潜在辅助治疗选择。
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Running and swimming prevent the deregulation of the BDNF/TrkB neurotrophic signalling at the neuromuscular junction in mice with amyotrophic lateral sclerosis.跑步和游泳可防止肌萎缩侧索硬化症小鼠神经肌肉接点处 BDNF/TrkB 神经营养信号的失调。
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1
Functional over-load saves motor units in the SOD1-G93A transgenic mouse model of amyotrophic lateral sclerosis.功能过载可拯救 SOD1-G93A 转基因肌萎缩侧索硬化症小鼠模型中的运动神经元。
Neurobiol Dis. 2010 Feb;37(2):412-22. doi: 10.1016/j.nbd.2009.10.021. Epub 2009 Oct 29.
2
Motoneuron survival is promoted by specific exercise in a mouse model of amyotrophic lateral sclerosis.在肌萎缩侧索硬化症小鼠模型中，特定运动可促进运动神经元存活。
J Physiol. 2009 Jul 15;587(Pt 14):3561-72. doi: 10.1113/jphysiol.2009.169748. Epub 2009 Jun 2.
3
Morphological differences between wild-type and transgenic superoxide dismutase 1 lumbar motoneurons in postnatal mice.出生后小鼠野生型和转基因超氧化物歧化酶1腰段运动神经元之间的形态学差异。
J Comp Neurol. 2008 Nov 20;511(3):329-41. doi: 10.1002/cne.21818.
4
Exercise-induced modulation of calcineurin activity parallels the time course of myofibre transitions.运动诱导的钙调神经磷酸酶活性调节与肌纤维转变的时间进程平行。
J Cell Physiol. 2008 Jan;214(1):126-35. doi: 10.1002/jcp.21168.
5
Early electrophysiological abnormalities in lumbar motoneurons in a transgenic mouse model of amyotrophic lateral sclerosis.肌萎缩侧索硬化转基因小鼠模型中腰段运动神经元的早期电生理异常
Eur J Neurosci. 2007 Jan;25(2):451-9. doi: 10.1111/j.1460-9568.2007.05306.x.
6
Effects of high-intensity endurance exercise training in the G93A mouse model of amyotrophic lateral sclerosis.高强度耐力运动训练对肌萎缩侧索硬化症G93A小鼠模型的影响。
Muscle Nerve. 2004 May;29(5):656-62. doi: 10.1002/mus.20004.
7
Amyotrophic lateral sclerosis is a distal axonopathy: evidence in mice and man.肌萎缩侧索硬化是一种远端轴索性神经病：来自小鼠和人类的证据。
Exp Neurol. 2004 Feb;185(2):232-40. doi: 10.1016/j.expneurol.2003.10.004.
8
Overexpression of HGF retards disease progression and prolongs life span in a transgenic mouse model of ALS.在肌萎缩侧索硬化症的转基因小鼠模型中，肝细胞生长因子（HGF）的过表达可延缓疾病进展并延长寿命。
J Neurosci. 2002 Aug 1;22(15):6537-48. doi: 10.1523/JNEUROSCI.22-15-06537.2002.
9
Adenovirus-mediated gene transfer of glial cell line-derived neurotrophic factor prevents motor neuron loss of transgenic model mice for amyotrophic lateral sclerosis.腺病毒介导的胶质细胞源性神经营养因子基因转移可预防肌萎缩侧索硬化转基因模型小鼠的运动神经元丧失。
Apoptosis. 2002 Aug;7(4):329-34. doi: 10.1023/a:1016123413038.
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Increased survival and function of SOD1 mice after glial cell-derived neurotrophic factor gene therapy.胶质细胞源性神经营养因子基因治疗后SOD1小鼠的存活率和功能提高。
Hum Gene Ther. 2002 Jun 10;13(9):1047-59. doi: 10.1089/104303402753812458.

The Effect of Training on Motoneuron Survival in Amyotrophic Lateral Sclerosis: Which Motoneuron Type is Saved?

作者信息

Elbasiouny Sherif M, Schuster Jenna E

机构信息

Heckman Lab, Department of Physiology, Feinberg School of Medicine, Northwestern University Chicago, IL, USA.

出版信息

Front Physiol. 2011 Apr 25;2:18. doi: 10.3389/fphys.2011.00018. eCollection 2011.

DOI:10.3389/fphys.2011.00018
PMID:21541255
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3083714/
Abstract
摘要