人HL-60白血病细胞核中微粒体激活以及4'-(9-吖啶基氨基)-3-甲磺酰基间氨基苯甲醚（m-AMSA）依赖性、拓扑异构酶相关DNA损伤的产生增加。 - Suppr

Microsomal activation and increased production of 4'-(9-acridinylamino)-3-methanesulfon-m-anisidide (m-AMSA)-dependent, topoisomerase-associated DNA lesions in nuclei from human HL-60 leukemia cells.

Gorsky L D, Morin M J

Department of Pharmacology, Northwestern University, Chicago, IL 60611.

Biochem Pharmacol. 1990 May 1;39(9):1481-4. doi: 10.1016/0006-2952(90)90430-s.

Abstract

摘要

Biochem Pharmacol. 1990 May 1;39(9):1481-4. doi: 10.1016/0006-2952(90)90430-s.

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