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中枢而非外周阿片受体阻断可延长垂体-肾上腺对应激的反应。

Central but not peripheral opiate receptor blockade prolonged pituitary-adrenal responses to stress.

作者信息

Odio M, Brodish A

机构信息

Department of Physiology and Pharmacology, Bowman Gray School of Medicine, Wake Forest University, Winston-Salem, NC 27103.

出版信息

Pharmacol Biochem Behav. 1990 Apr;35(4):963-9. doi: 10.1016/0091-3057(90)90386-v.

Abstract

Evidence from pharmacological studies suggest that opiate systems may serve either inhibitory or stimulatory functions on stress-induced responses of the hypothalamic-pituitary-adrenocortical (HPA) axis. The objective of these experiments was to determine whether these discrepant findings may result, in part, from differential effects of central or peripheral opiate receptor blockade on HPA axis responses. To this effect, groups of rats received injections of either saline, naltrexone (NHCl) or the quaternary analogue naltrexone methobromide (NMBr). The animals were then exposed to 30 min of a motion stressor and blood samples were obtained from each rat for analysis of ACTH, corticosterone, and prolactin. The data showed that resting and stress-induced levels of prolactin were decreased by NHCl only. Although neither drug affected the magnitude of the stress-induced ACTH and corticosterone responses, treatment with NHCl, but not NMBr, delayed the poststress decline of these responses. Hence, we concluded that central opiate mechanisms may be important for cessation of HPA axis activity, after exposure to stressful situations.

摘要

药理学研究的证据表明,阿片系统可能对下丘脑 - 垂体 - 肾上腺皮质(HPA)轴的应激诱导反应发挥抑制或刺激作用。这些实验的目的是确定这些相互矛盾的发现是否部分源于中枢或外周阿片受体阻断对HPA轴反应的不同影响。为此,将大鼠分组,分别注射生理盐水、纳曲酮(NHCl)或季铵类似物纳曲酮甲溴化物(NMBr)。然后让动物暴露于30分钟的运动应激源,从每只大鼠采集血样以分析促肾上腺皮质激素(ACTH)、皮质酮和催乳素。数据显示,仅NHCl降低了静息和应激诱导的催乳素水平。虽然两种药物均未影响应激诱导的ACTH和皮质酮反应的幅度,但NHCl治疗而非NMBr治疗延迟了应激后这些反应的下降。因此,我们得出结论,中枢阿片机制可能在暴露于应激情况后对HPA轴活动的停止起重要作用。

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