Beliakov N A, Pleukhova O A, Servatinskiĭ G L
Vestn Khir Im I I Grek. 1990 Aug;145(8):58-62.
An analysis of 2049 autopsies and experimental investigations in 150 albino rats have shown that pulmonary complications developing during the first two weeks after cranio-cerebral traumas include pulmonary edema, aspiration syndrome, pneumonia. The pulmonary injuries are based on several pathogenetic mechanisms associated with the effect of hemodynamics, humoral factors, activation of leukocytes, aspiration and infection. Disturbed hemodynamics and vascular permeability in the lungs can be due to functional alterations in the zone of vascular-motor and respiratory centers.
对2049例尸检以及对150只白化大鼠进行的实验研究分析表明，颅脑创伤后前两周出现的肺部并发症包括肺水肿、吸入综合征、肺炎。肺部损伤基于几种与血流动力学、体液因素、白细胞活化、吸入和感染作用相关的发病机制。肺部血流动力学和血管通透性紊乱可能是由于血管运动和呼吸中枢区域的功能改变所致。
