Végh Eszter Mária, Széplaki Gábor, Szilágyi Szabolcs, Osztheimer István, Tahin Tamás, Merkely Béla, Gellér László
Semmelweis Egyetem, Általános Orvostudományi Kar, Kardiológiai Központ, Budapest.
Orv Hetil. 2011 Aug 21;152(34):1374-8. doi: 10.1556/OH.2011.29194.
A 29-year-old male was admitted to our outpatient clinic because of palpitation and documented narrow QRS arrhythmia. Based on the ECG, supraventricular tachycardia was diagnosed, electrophysiological examination was indicated and ablation therapy was recommended. During positioning of the catheter the patient developed arrhythmia. On the coronary sinus catheter the activation spread from distal to proximal electrodes, suggesting left atrial origin. During atrial entrainment pacing long return cycle was observed and distal coronary sinus pacing resulted in a 15 ms longer cycle length than the arrhythmia. Therefore, the left atrial origin of the arrhythmia was confirmed and double transseptal puncture was performed. Lasso and irrigated tip catheter were introduced into the left atrium and electroanatomical mapping was performed with CARTO3 system. After electroanatomical mapping the origin of tachycardia was located proximally in the left superior pulmonary vein. Ablation was started at the earliest activation point, where acceleration was observed and the arrhythmia stopped after the first ablation. Pulmonary vein isolation was completed, and bidirectional block could be confirmed. After 30 minutes the arrhythmia was not inducible. During follow-up, Holter-examination was negative and the patient remained asymptomatic. The pulmonary vein tachycardia is a supraventricular arrhythmia that can occur at any age, but the diagnosis based on the ECG is not always simple. Detailed electroanatomical mapping is very important in the diagnosis of this type of arrhythmia, although it can be verified with conventional electrophysiological methods as well. Focal ablation may be a therapeutic option; however, total isolation of pulmonary veins can be more effective.
一名29岁男性因心悸及记录到的窄QRS波心律失常入住我院门诊。根据心电图诊断为室上性心动过速,建议进行电生理检查及消融治疗。在导管定位过程中,患者出现心律失常。在冠状窦导管上,激动从远端电极向近端电极传播,提示起源于左心房。心房拖带起搏时观察到长回归周期,冠状窦远端起搏导致周期长度比心律失常时长15毫秒。因此,心律失常的左心房起源得以证实,并进行了双房间隔穿刺。将Lasso和灌注射频消融导管送入左心房,并用CARTO3系统进行电解剖标测。电解剖标测后,心动过速起源位于左上肺静脉近端。在最早出现激动且观察到加速的部位开始消融,首次消融后心律失常终止。完成肺静脉隔离,并证实双向阻滞。30分钟后,心律失常不能被诱发。随访期间,动态心电图检查结果为阴性,患者仍无症状。肺静脉性心动过速是一种可发生于任何年龄的室上性心律失常,但基于心电图的诊断并不总是简单的。详细的电解剖标测在这类心律失常的诊断中非常重要,尽管也可用传统电生理方法进行验证。局灶性消融可能是一种治疗选择;然而,肺静脉完全隔离可能更有效。