Levine R A, Wolff D A
Intervirology. 1979;11(4):255-60. doi: 10.1159/000149042.
When bovine enterovirus (BEV) RNA synthesis was inhibited by 2-(alpha-hydroxybenzyl)-benzimidazole (HBB), an increase in MOI was accompanied by an enhancement of cytopathology. Since BEV RNA synthesis is inhibited and BEV protein synthesis is allowed to proceed in the presence of HBB, these results support the concept that proteins play a role in the development of cytopathology independently of viral double-stranded RNA.
当牛肠道病毒（BEV）的RNA合成被2-（α-羟基苄基）-苯并咪唑（HBB）抑制时，感染复数（MOI）的增加伴随着细胞病变的增强。由于在HBB存在的情况下BEV的RNA合成被抑制而BEV蛋白合成仍可进行，这些结果支持了蛋白质在细胞病变发展中发挥作用且独立于病毒双链RNA的观点。
