Green R M, Kelly K M, Gabrielsen T, Levine S R, Vanderzant C
Department of Neurology, University of Michigan Medical Center, Ann Arbor.
Stroke. 1990 Jun;21(6):957-62. doi: 10.1161/01.str.21.6.957.
After smoking "crack" cocaine and consuming large quantities of ethanol, a 36-year-old man developed multiple, bilateral, deep, and superficial cerebral hematomas. He was hypertensive for several days, but angiography revealed no evidence of vascular malformation or vasculitis. The multifocality of the hematomas and lack of underlying disease suggest that the hemorrhages resulted from cocaine-induced acute hypertension or arterial spasm, possibly potentiated by heavy ethanol consumption.
一名36岁男子在吸食“快克”可卡因并大量饮用乙醇后,出现了多发性、双侧性、深部和浅部脑血肿。他高血压持续了几天,但血管造影未显示血管畸形或血管炎的证据。血肿的多灶性以及无潜在疾病表明,出血是由可卡因引起的急性高血压或动脉痉挛所致,大量饮用乙醇可能起到了增强作用。