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Glial Cell Line-Derived Neurotrophic Factor Gene Delivery in Parkinson's Disease: A Delicate Balance between Neuroprotection, Trophic Effects, and Unwanted Compensatory Mechanisms.
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Ret is essential to mediate GDNF's neuroprotective and neuroregenerative effect in a Parkinson disease mouse model.
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RET revisited: expanding the oncogenic portfolio.
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The proform of glia cell line-derived neurotrophic factor: a potentially biologically active protein.
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Functional neuroprotection and efficient regulation of GDNF using destabilizing domains in a rodent model of Parkinson's disease.
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Castration induces Parkinson disease pathologies in young male mice via inducible nitric-oxide synthase.
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Molecular mechanisms of RET receptor-mediated oncogenesis in multiple endocrine neoplasia 2.
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GDNF fails to exert neuroprotection in a rat α-synuclein model of Parkinson's disease.
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Induction of the unfolded protein response by α-synuclein in experimental models of Parkinson's disease.
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Gene expression patterns for GDNF and its receptors in the human putamen affected by Parkinson's disease: a real-time PCR study.
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Expression of the proto-oncogene Ret, a component of the GDNF receptor complex, persists in human substantia nigra neurons in Parkinson's disease.
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Peripheral expression and biological activities of GDNF, a new neurotrophic factor for avian and mammalian peripheral neurons.
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Glial cell line-derived neurotrophic factor augments midbrain dopaminergic circuits in vivo.
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