Shurandina N V
Med Radiol (Mosk). 1990 Jul;35(7):29-32.
Ultrasound investigation of the abdominal vessels was conducted in 41 patients with liver tumors (17 patients with liver hemangiomas, 9 with primary liver cancer, and 15 with cancer metastases to the liver) and 100 controls. Shifting, compression, occlusion of the intrahepatic portal and hepatic veins as well as of the inferior vena cava and right renal vessels were observed in patients with malignant and benign liver tumors. Malignant tumors were accountable for shifting and compression of the extrahepatic segment of the portal vein and the upper mesenteric artery as a result of metastatic involvement of the lymph nodes. Statistically significant dilatation of the total hepatic artery was noted in patients with malignant and benign liver tumors, determined, probably, by an increase in the arterial inflow in the liver. A decrease in a degree of change of calibers of the portal and hepatic veins in malignant liver tumors was noted in patients with hepatocellular cancer, developing against a background of cirrhosis with portal hypertension as well as in patients with secondary malignant tumors without clinical signs of portal hypertension that might result from an obstacle posed by tumor nodes in the liver parenchyma to venous wall dilatation.
对41例肝肿瘤患者（17例肝血管瘤、9例原发性肝癌、15例肝癌转移）和100例对照者进行了腹部血管超声检查。在恶性和良性肝肿瘤患者中均观察到肝内门静脉、肝静脉以及下腔静脉和右肾血管的移位、受压和闭塞。恶性肿瘤导致肝外门静脉段和肠系膜上动脉移位和受压是由于淋巴结转移累及所致。在恶性和良性肝肿瘤患者中均观察到肝总动脉有统计学意义的扩张，这可能是由于肝脏动脉血流增加所致。在伴有门静脉高压的肝硬化背景下发生的肝细胞癌患者以及无门静脉高压临床体征的继发性恶性肿瘤患者中，观察到恶性肝肿瘤患者门静脉和肝静脉管径变化程度降低，这可能是由于肝实质内肿瘤结节对静脉壁扩张造成阻碍所致。
