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1
Nucleotide sequence of the psiA (plasmid SOS inhibition) gene located on the leading region of plasmids F and R6-5.位于质粒F和R6 - 5前导区域的psiA（质粒SOS抑制）基因的核苷酸序列。
Nucleic Acids Res. 1990 Aug 11;18(15):4597. doi: 10.1093/nar/18.15.4597.
2
Identification of psiB genes of plasmids F and R6-5. Molecular basis for psiB enhanced expression in plasmid R6-5.质粒F和R6 - 5的psiB基因鉴定。psiB在质粒R6 - 5中增强表达的分子基础。
Nucleic Acids Res. 1988 Nov 25;16(22):10669-79. doi: 10.1093/nar/16.22.10669.
3
A gene encoding an SOS inhibitor is present in different conjugative plasmids.一种编码SOS抑制剂的基因存在于不同的接合质粒中。
J Bacteriol. 1988 Sep;170(9):4392-4. doi: 10.1128/jb.170.9.4392-4394.1988.
4
Sequence and conservation of genes at the distal end of the transfer region on plasmids F and R6-5.质粒F和R6 - 5转移区域远端基因的序列与保守性。
Gene. 1991 Jul 31;104(1):85-90. doi: 10.1016/0378-1119(91)90469-r.
5
pR plasmid replication provides evidence that single-stranded DNA induces the SOS system in vivo.pR质粒复制证明单链DNA在体内可诱导SOS系统。
Mol Gen Genet. 1993 Apr;238(3):333-8. doi: 10.1007/BF00291991.
6
The recQ gene of Escherichia coli K12: primary structure and evidence for SOS regulation.大肠杆菌K12的recQ基因：一级结构及SOS调控的证据
Mol Gen Genet. 1986 Nov;205(2):298-304. doi: 10.1007/BF00430442.
7
Repressor gene finO in plasmids R100 and F: constitutive transfer of plasmid F is caused by insertion of IS3 into F finO.质粒R100和F中的阻遏基因finO：质粒F的组成型转移是由IS3插入F finO引起的。
J Bacteriol. 1987 Feb;169(2):619-23. doi: 10.1128/jb.169.2.619-623.1987.
8
PsiB, and anti-SOS protein, is transiently expressed by the F sex factor during its transmission to an Escherichia coli K-12 recipient.PsiB，一种抗SOS蛋白，在F性因子传递给大肠杆菌K-12受体的过程中短暂表达。
Mol Microbiol. 1992 Apr;6(7):885-93. doi: 10.1111/j.1365-2958.1992.tb01539.x.
9
Nucleotide sequence of the F plasmid leading region.
Plasmid. 1999 May;41(3):219-25. doi: 10.1006/plas.1999.1390.
10
[Bioluminescent analysis of the SOS-response of Escherichia coli cells].[大肠杆菌细胞SOS反应的生物发光分析]
Genetika. 1996 Mar;32(3):354-8.

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1
Real-time visualisation of the intracellular dynamics of conjugative plasmid transfer.实时可视化共轭质粒转移的细胞内动力学。
Nat Commun. 2023 Jan 18;14(1):294. doi: 10.1038/s41467-023-35978-3.
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Complete DNA sequence of a ColBM plasmid from avian pathogenic Escherichia coli suggests that it evolved from closely related ColV virulence plasmids.来自禽致病性大肠杆菌的ColBM质粒的完整DNA序列表明，它是由密切相关的ColV毒力质粒进化而来的。
J Bacteriol. 2006 Aug;188(16):5975-83. doi: 10.1128/JB.00204-06.
3
DNA sequence of a ColV plasmid and prevalence of selected plasmid-encoded virulence genes among avian Escherichia coli strains.一种ColV质粒的DNA序列以及禽源大肠杆菌菌株中选定的质粒编码毒力基因的流行情况。
J Bacteriol. 2006 Jan;188(2):745-58. doi: 10.1128/JB.188.2.745-758.2006.
4
DNA sequence and comparative genomics of pAPEC-O2-R, an avian pathogenic Escherichia coli transmissible R plasmid.禽致病性大肠杆菌可传播的R质粒pAPEC-O2-R的DNA序列及比较基因组学
Antimicrob Agents Chemother. 2005 Nov;49(11):4681-8. doi: 10.1128/AAC.49.11.4681-4688.2005.
5
Analysis of the sequence and gene products of the transfer region of the F sex factor.F 性因子转移区域的序列及基因产物分析
Microbiol Rev. 1994 Jun;58(2):162-210. doi: 10.1128/mr.58.2.162-210.1994.
6
New nucleotide sequence data on the EMBL File Server.欧洲分子生物学实验室文件服务器上的新核苷酸序列数据。
Nucleic Acids Res. 1990 Oct 25;18(20):6181-92. doi: 10.1093/nar/18.20.6181.

本文引用的文献

1
Cloning and analysis of pif, replication and leading regions of the F plasmid.F质粒的pif、复制和前导区域的克隆与分析
Mol Gen Genet. 1984;197(1):129-36. doi: 10.1007/BF00327933.
2
An inhibitor of SOS induction, specified by a plasmid locus in Escherichia coli.一种由大肠杆菌中的质粒位点所确定的SOS诱导抑制剂。
Proc Natl Acad Sci U S A. 1986 Aug;83(15):5723-6. doi: 10.1073/pnas.83.15.5723.
3
PsiB polypeptide prevents activation of RecA protein in Escherichia coli.PsiB多肽可防止大肠杆菌中RecA蛋白的激活。
Mol Gen Genet. 1988 Nov;214(3):389-95. doi: 10.1007/BF00330471.

Nucleotide sequence of the psiA (plasmid SOS inhibition) gene located on the leading region of plasmids F and R6-5.

作者信息

Loh S, Skurray R, Célérier J, Bagdasarian M, Bailone A, Devoret R

机构信息

Department of Microbiology, Monash University, Clayton, Victoria, Australia.

出版信息

Nucleic Acids Res. 1990 Aug 11;18(15):4597. doi: 10.1093/nar/18.15.4597.

DOI:10.1093/nar/18.15.4597
PMID:2201950
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC331290/
Abstract
摘要