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SCH58261 是一种选择性的腺苷 A(2A)受体阻断剂,可调节双侧颈总动脉闭塞后的缺血再灌注损伤:炎症介质的作用。

SCH58261 the selective adenosine A(2A) receptor blocker modulates ischemia reperfusion injury following bilateral carotid occlusion: role of inflammatory mediators.

机构信息

Department of Pharmacology and Toxicology, Faculty of Pharmacy, Cairo University, Kasr El-Aini Street, Cairo 11562, Egypt.

出版信息

Neurochem Res. 2012 Mar;37(3):538-47. doi: 10.1007/s11064-011-0640-x. Epub 2011 Nov 10.

DOI:10.1007/s11064-011-0640-x
PMID:22071908
Abstract

In the present study, the effects of SCH58261, a selective adenosine A(2A) receptor antagonist that crosses the blood brain barrier (BBB) and 8-(4-sulfophenyl) theophylline (8-SPT), a non-selective adenosine receptor antagonist that acts peripherally, were investigated on cerebral ischemia reperfusion injury (IR). Male Wistar rats (200-250 g) were divided into four groups: (1) sham-operated (SO), IR pretreated with either (2) vehicle (DMSO); (3) SCH58261 (0.01 mg/kg); (4) 8-SPT (2.5 mg/kg). Animals were anesthetized and submitted to occlusion of both carotid arteries for 45 min. All treatments were administered intraperitoneally (i.p.) post carotid occlusion prior to exposure to a 24 h reperfusion period. Ischemic rats showed increased infarct size compared to their control counterparts that corroborated with histopathological changes as well as increased lactate dehydrogenase (LDH) activity in the hippocampus. Moreover, ischemic animals showed habituation deficit, increased anxiety and locomotor activity. IR increased hippocampal glutamate (Glu), GABA, glycine (Gly) and aspartate (ASP). SCH58261 significantly reversed these effects while 8-SPT elicited minimal change. IR raised myeloperoxidase (MPO), tumor necrosis factor-alpha (TNF-α), nitric oxide (NO), prostaglandin E₂ (PGE₂) accompanied by a decrease in interleukin-10 (IL-10), effects that were again reversed by SCH58261, but 8-SPT elicited less changes. Results from the present study point towards the importance of central blockade of adenosine A(2A) receptor in ameliorating hippocampal damage following IR injury by halting inflammatory cascades as well as modulating excitotoxicity.

摘要

在本研究中,研究了 SCH58261(一种可穿透血脑屏障(BBB)的选择性腺苷 A(2A)受体拮抗剂)和 8-(4-磺苯基)茶碱(8-SPT,一种具有外周作用的非选择性腺苷受体拮抗剂)对脑缺血再灌注损伤(IR)的影响。雄性 Wistar 大鼠(200-250g)分为四组:(1)假手术(SO),IR 预处理用(2)载体(DMSO);(3)SCH58261(0.01mg/kg);(4)8-SPT(2.5mg/kg)。动物麻醉后,夹闭双侧颈总动脉 45 分钟。所有治疗均在颈总动脉闭塞后腹腔内(ip)给予,然后暴露于 24 小时再灌注期。与对照组相比,缺血大鼠的梗死面积增加,与组织病理学变化以及海马中乳酸脱氢酶(LDH)活性的增加相吻合。此外,缺血动物表现出习惯化缺陷、焦虑和运动活动增加。IR 增加了海马中的谷氨酸(Glu)、γ-氨基丁酸(GABA)、甘氨酸(Gly)和天冬氨酸(ASP)。SCH58261 显著逆转了这些作用,而 8-SPT 则引起了最小的变化。IR 增加了髓过氧化物酶(MPO)、肿瘤坏死因子-α(TNF-α)、一氧化氮(NO)、前列腺素 E₂(PGE₂),同时白细胞介素-10(IL-10)减少,这些作用再次被 SCH58261 逆转,但 8-SPT 引起的变化较小。本研究的结果表明,通过阻断炎症级联反应和调节兴奋性毒性,中枢阻断腺苷 A(2A)受体对改善 IR 损伤后海马损伤具有重要意义。

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