细胞骨架成分在细胞松弛素E诱导人多形核白细胞产生超氧化物中的作用
Role of cytoskeletal elements in cytochalasin E-induced superoxide production by human polymorphonuclear leukocytes.
作者信息
Nakagawara A, Minakami S
出版信息
Biochim Biophys Acta. 1979 Apr 18;584(1):143-8. doi: 10.1016/0304-4165(79)90244-7.
PMID:221046
Abstract
The release of superoxide anions from human polymorphonuclear leukocytes induced by cytochalasin E was greatly enhanced by the pretreatment of the cells either with deuterium oxide or with concanavalin A. Colchicine, vinblastine and cyclic AMP inhibited the release. Cytochalasins A and B also suppressed the superoxide release. These observations suggest the involvement of microfilament-microtubule system in the production and release of superoxide anions induced by cytochalasin E.
摘要
用细胞松弛素E诱导人多形核白细胞释放超氧阴离子，若先用氧化氘或伴刀豆球蛋白A预处理细胞，则超氧阴离子的释放会大大增强。秋水仙碱、长春碱和环磷酸腺苷可抑制其释放。细胞松弛素A和B也可抑制超氧阴离子的释放。这些观察结果表明，微丝-微管系统参与了细胞松弛素E诱导的超氧阴离子的产生和释放。
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