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The clinical characteristics and pathogenic variants of primary pigmented nodular adrenocortical disease in 210 patients: a systematic review.
Front Endocrinol (Lausanne). 2024 Jun 26;15:1356870. doi: 10.3389/fendo.2024.1356870. eCollection 2024.
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Pediatric adrenocortical carcinoma.
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Bridging the Scientific Gaps to Identify Effective Treatments in Adrenocortical Cancer.
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Pathological and Genetic Stratification for Management of Adrenocortical Carcinoma.
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Current Status and Future Targeted Therapy in Adrenocortical Cancer.
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本文引用的文献

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β-catenin activation is associated with specific clinical and pathologic characteristics and a poor outcome in adrenocortical carcinoma.
Clin Cancer Res. 2011 Jan 15;17(2):328-36. doi: 10.1158/1078-0432.CCR-10-2006. Epub 2010 Nov 18.
4
Association of the M1V PRKAR1A mutation with primary pigmented nodular adrenocortical disease in two large families.
J Clin Endocrinol Metab. 2010 Jan;95(1):338-42. doi: 10.1210/jc.2009-0993. Epub 2009 Nov 13.
6
New genes and/or molecular pathways associated with adrenal hyperplasias and related adrenocortical tumors.
Mol Cell Endocrinol. 2009 Mar 5;300(1-2):152-7. doi: 10.1016/j.mce.2008.11.010. Epub 2008 Nov 21.
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Protein kinase A effects of an expressed PRKAR1A mutation associated with aggressive tumors.
Cancer Res. 2008 May 1;68(9):3133-41. doi: 10.1158/0008-5472.CAN-08-0064.
9
Detection of somatic beta-catenin mutations in primary pigmented nodular adrenocortical disease (PPNAD).
Clin Endocrinol (Oxf). 2008 Sep;69(3):367-73. doi: 10.1111/j.1365-2265.2008.03273.x. Epub 2008 Apr 14.

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