Department of Anesthesiology and Intensive Care, Sainte Anne Teaching Military Hospital, Toulon, France.
Neurocrit Care. 2012 Feb;16(1):145-7. doi: 10.1007/s12028-011-9655-z.
Cerebral vasospasm is the main cause of neurological mortality and morbidity following subarachnoid hemorrhage. Basilar artery vasospasm (BAVS) is associated with a high morbidity and may have multiple clinical presentations.
We report the case of a 43 years-old man with BAVS presenting as a reversible locked-in syndrome (LIS) after stopping sedation.
The symptoms were successfully managed by intra-arterial infusion of vasodilators and balloon angioplasty. Magnetic resonance imaging did not reveal any brainstem lesion 48 h after the complication, demonstrating a hemodynamic mechanism.
LIS can reveal BAVS. Its diagnosis relies on clinical examination. In this case, rapid neuro-interventional treatment permitted reversal of symptoms. This could not have been possible under sedation.
蛛网膜下腔出血后,脑血管痉挛是导致神经功能死亡率和发病率的主要原因。基底动脉痉挛(BAVS)与高发病率相关,可能有多种临床表现。
我们报告了一例 43 岁男性患者,在停止镇静后表现为可逆性闭锁综合征(LIS),伴有 BAVS。
通过动脉内输注血管扩张剂和球囊血管成形术成功治疗了症状。并发症发生后 48 小时,磁共振成像未显示任何脑干病变,表明存在血流动力学机制。
LIS 可揭示 BAVS。其诊断依赖于临床检查。在这种情况下,快速神经介入治疗可逆转症状。在镇静下这是不可能的。