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两兄弟溃疡性结肠炎巯嘌呤代谢异常,经别嘌醇和巯嘌呤剂量减少治疗后成功缓解。

Two brothers with skewed thiopurine metabolism in ulcerative colitis treated successfully with allopurinol and mercaptopurine dose reduction.

机构信息

Inflammatory Bowel Disease Center, University of Chicago, Chicago, IL, USA.

出版信息

Dig Dis Sci. 2012 Jan;57(1):250-3. doi: 10.1007/s10620-011-1999-x. Epub 2011 Dec 7.

Abstract

Thiopurine therapy effectively maintains remission in inflammatory bowel disease. However, many patients are unable to achieve optimum benefits from azathioprine or 6-mercaptopurine because of undesirable metabolism related to high thiopurine methyltransferase (TPMT) activity characterized by hepatic transaminitis secondary to increased 6-methylmercaptopurine (6-MMP) production and reduced levels of therapeutic 6-thioguanine nucleotide (6-TGN). Allopurinol can optimize this skewed metabolism. We discuss two brothers who were both diagnosed with ulcerative colitis (UC). Their disease remained active despite oral and topical mesalamines. Steroids followed by 6-mercaptopurine (MP) were unsuccessfully introduced for both patients and both were found to have high 6-MMP and low 6-TGN levels, despite normal TMPT enzyme activity, accompanied by transaminitis. Allopurinol was introduced in combination with MP dose reduction. For both brothers addition of allopurinol was associated with successful remission and optimized MP metabolites. These siblings with active UC illustrate that skewed thiopurine metabolism may occur despite normal TPMT enzyme activity and can lead to adverse events in the absence of disease control. We confirm previous data showing that addition of allopurinol can reverse this skewed metabolism, and reduce both hepatotoxicity and disease activity, but we now also introduce the concept of a family history of preferential MP metabolism as a clue to effective management for other family members.

摘要

硫嘌呤治疗能有效地维持炎症性肠病的缓解。然而,由于与高硫嘌呤甲基转移酶(TPMT)活性相关的不良代谢,许多患者无法从硫唑嘌呤或 6-巯基嘌呤中获得最佳疗效,其特征为由于增加的 6-甲基巯基嘌呤(6-MMP)产生和降低的治疗性 6-硫鸟嘌呤核苷酸(6-TGN)水平导致肝转氨基酶升高。别嘌醇可以优化这种代谢失衡。我们讨论了两位均被诊断为溃疡性结肠炎(UC)的兄弟。尽管使用了口服和局部美沙拉嗪,他们的疾病仍处于活动期。为这两位患者都尝试了引入类固醇,然后是 6-巯基嘌呤(MP),但均未成功,尽管 TPMT 酶活性正常,但都发现 6-MMP 水平升高和 6-TGN 水平降低,伴有转氨基酶升高。联合使用别嘌醇和减少 MP 剂量。对于这两兄弟,添加别嘌醇都与成功缓解和优化 MP 代谢物有关。这对患有活动性 UC 的兄弟表明,尽管 TPMT 酶活性正常,但仍可能发生硫嘌呤代谢失衡,并导致在未控制疾病的情况下出现不良反应。我们证实了之前的数据,表明添加别嘌醇可以逆转这种代谢失衡,并降低肝毒性和疾病活动度,但我们现在还引入了家族中优先 MP 代谢的病史作为其他家族成员有效管理的线索。

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