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The structural proteins of murine leukemia virus and the pathogenesis of necrotizing arteritis and glomerulonephritis in SL/Ni mice.

作者信息

Yoshiki T, Hayasaka T, Fukatsu R, Shirai T, Itoh T, Ikeda H, Katagiri M

出版信息

J Immunol. 1979 May;122(5):1812-20.

PMID:221579
Abstract
摘要

相似文献

1
The structural proteins of murine leukemia virus and the pathogenesis of necrotizing arteritis and glomerulonephritis in SL/Ni mice.
J Immunol. 1979 May;122(5):1812-20.
2
[C-type murine leukemia virus and the pathogenesis of necrotizing arteritis and glomerulonephritis in MRL/1 mice].[C型鼠白血病病毒与MRL/1小鼠坏死性动脉炎和肾小球肾炎的发病机制]
Hokkaido Igaku Zasshi. 1982 Nov;57(6):688-95.
3
Specificity of cytolytic T cells directed against AKR/Gross virus-induced syngeneic leukemias: antibodies directed against H-2K, but not against viral proteins, inhibit lysis.针对AKR/格罗斯病毒诱导的同基因白血病的细胞溶解性T细胞的特异性：针对H-2K而非病毒蛋白的抗体可抑制细胞溶解。
J Immunol. 1980 Aug;125(2):647-55.
4
Spontaneous necrotizing arteritis and glomerulonephritis in mice.小鼠自发性坏死性动脉炎和肾小球肾炎
Nihon Jinzo Gakkai Shi. 1981 Jul;23(7):931-8.
5
The GIX antigen of murine leukemia virus: an analysis with monoclonal antibodies.鼠白血病病毒的GIX抗原：用单克隆抗体进行的分析
Virology. 1981 Jul 30;112(2):450-60. doi: 10.1016/0042-6822(81)90292-0.
6
Immunopathogenicity and oncogenicity of murine leukemia viruses. II. Infection of mice and rats with Scripps leukemia virus.鼠白血病病毒的免疫致病性和致癌性。II. 用斯克里普斯白血病病毒感染小鼠和大鼠。
Am J Pathol. 1976 Feb;82(2):299-314.
7
Serological analysis of cell surface antigens of tumors induced by murine leukemia virus.鼠白血病病毒诱导的肿瘤细胞表面抗原的血清学分析。
J Natl Cancer Inst. 1972 Jan;48(1):265-71.
8
Group-specific cytolytic antibody directed against the major glycoprotein (gp70) of murine leukemia viruses in serum of mice with dormant FLV infections.针对潜伏性FLV感染小鼠血清中鼠白血病病毒主要糖蛋白（gp70）的组特异性溶细胞抗体。
Virology. 1979 Aug;97(1):55-67. doi: 10.1016/0042-6822(79)90372-6.
9
The specificity of H-2-restricted cytotoxic T lymphocytes directed to AKR/Gross leukemia virus-induced tumors. III. Coordinate alterations in viral gp70 antigen expression and restoration of CTL-susceptibility to insusceptible variant tumors.针对AKR/格罗斯白血病病毒诱导肿瘤的H-2限制性细胞毒性T淋巴细胞的特异性。III. 病毒gp70抗原表达的协同改变以及细胞毒性T淋巴细胞对不敏感变异肿瘤敏感性的恢复
J Immunol. 1986 Mar 15;136(6):2271-9.
10
Properties of mouse leukemia viruses. XVII. Factors required for successful treatment of spontaneous AKR leukemia by antibodies against gp71.小鼠白血病病毒的特性。十七。用抗gp71抗体成功治疗自发性AKR白血病所需的因素。
Virology. 1981 Jun;111(2):568-78. doi: 10.1016/0042-6822(81)90358-5.

引用本文的文献

1
Pathogenesis of Vasculitis in env-pX Rats.env-pX大鼠血管炎的发病机制
Ann Vasc Dis. 2012;5(3):296-9. doi: 10.3400/avd.ra.12.00063.
2
Tissue specific high level expression of a full length human endogenous retrovirus genome transgene, HERV-R, under control of its own promoter in rats.在大鼠中，全长人类内源性逆转录病毒基因组转基因HERV-R在其自身启动子的控制下进行组织特异性高水平表达。
Transgenic Res. 2003 Jun;12(3):319-28. doi: 10.1023/a:1023381819572.
3
Genetic regulation of the class conversion of dsDNA-specific antibodies in (NZB X NZW)F1 hybrid.
（NZB×NZW）F1 杂交小鼠中双链 DNA 特异性抗体类别转换的遗传调控
Immunogenetics. 1983;18(5):513-24. doi: 10.1007/BF00364392.
4
Autoimmune disease and malignant lymphoma associated with host-versus-graft disease in mice.与小鼠宿主抗移植物病相关的自身免疫性疾病和恶性淋巴瘤。
Clin Exp Immunol. 1985 Dec;62(3):535-44.
5
Pathogenesis of arteritis of SL/Ni mice. Possible lytic effect of anti-gp70 antibodies on vascular smooth muscle cells.SL/Ni小鼠动脉炎的发病机制。抗gp70抗体对血管平滑肌细胞可能的溶解作用。
J Exp Med. 1987 Oct 1;166(4):890-908. doi: 10.1084/jem.166.4.890.