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The importance of CELF control: molecular and biological roles of the CUG-BP, Elav-like family of RNA-binding proteins.
Wiley Interdiscip Rev RNA. 2012 Jan-Feb;3(1):104-21. doi: 10.1002/wrna.107. Epub 2011 Aug 17.
3
CUG-BP, Elav-like family (CELF)-mediated alternative splicing regulation in the brain during health and disease.
Mol Cell Neurosci. 2013 Sep;56:456-64. doi: 10.1016/j.mcn.2012.12.003. Epub 2012 Dec 14.
4
Cloning and embryonic expression patterns of the chicken CELF family.
Dev Dyn. 2007 Aug;236(8):2216-24. doi: 10.1002/dvdy.21209.
8
Identification of CELF splicing activation and repression domains in vivo.
Nucleic Acids Res. 2005 May 13;33(9):2769-80. doi: 10.1093/nar/gki561. Print 2005.
9
The role of CELF proteins in neurological disorders.
RNA Biol. 2010 Jul-Aug;7(4):474-9. doi: 10.4161/rna.7.4.12345. Epub 2010 Jul 1.
10
CUG-BP1/CELF1 requires UGU-rich sequences for high-affinity binding.
Biochem J. 2006 Dec 1;400(2):291-301. doi: 10.1042/BJ20060490.

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2
Adipocyte RNA-binding protein CELF1 promotes beiging of white fat through stabilizing Dio2 mRNA.
Nat Commun. 2025 Aug 11;16(1):7414. doi: 10.1038/s41467-025-62740-8.
4
Transition of CELF2 PAS usage promotes recovery of refractory JDM through alternative splicing regulation of CTSB.
Mol Ther Nucleic Acids. 2025 Jul 1;36(3):102624. doi: 10.1016/j.omtn.2025.102624. eCollection 2025 Sep 9.
6
CUGBP Elav-like family member 4 promotes cardiac remodeling through Inhibition of FMO2.
BMC Cardiovasc Disord. 2025 Jul 3;25(1):454. doi: 10.1186/s12872-025-04914-2.
7
Dysregulated RNA-binding proteins and alternative splicing: Emerging roles in autism spectrum disorder.
Mol Cells. 2025 Jun 3;48(8):100237. doi: 10.1016/j.mocell.2025.100237.
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Alternative splicing factors and cardiac disease: more than just missplicing?
RNA. 2025 Feb 19;31(3):300-306. doi: 10.1261/rna.080332.124.

本文引用的文献

1
HITS-CLIP: panoramic views of protein-RNA regulation in living cells.
Wiley Interdiscip Rev RNA. 2010 Sep-Oct;1(2):266-86. doi: 10.1002/wrna.31. Epub 2010 Aug 2.
3
Mis-splicing of Tau exon 10 in myotonic dystrophy type 1 is reproduced by overexpression of CELF2 but not by MBNL1 silencing.
Biochim Biophys Acta. 2011 Jul;1812(7):732-42. doi: 10.1016/j.bbadis.2011.03.010. Epub 2011 Mar 23.
4
Alternative splicing dysregulation secondary to skeletal muscle regeneration.
Ann Neurol. 2011 Apr;69(4):681-90. doi: 10.1002/ana.22278. Epub 2011 Mar 11.
6
Structural insights into RNA recognition by the alternate-splicing regulator CUG-binding protein 1.
Structure. 2010 Oct 13;18(10):1364-77. doi: 10.1016/j.str.2010.06.018.
7
Myotonic dystrophies 1 and 2: complex diseases with complex mechanisms.
Curr Genomics. 2010 Apr;11(2):77-90. doi: 10.2174/138920210790886844.
8
CELF and PTB proteins modulate the inclusion of the β-tropomyosin exon 6B during myogenic differentiation.
Exp Cell Res. 2011 Jan 1;317(1):94-106. doi: 10.1016/j.yexcr.2010.09.010. Epub 2010 Sep 25.
9
APOBEC-1-mediated RNA editing.
Wiley Interdiscip Rev Syst Biol Med. 2010 Sep-Oct;2(5):594-602. doi: 10.1002/wsbm.82.
10
BrunoL1 regulates endoderm proliferation through translational enhancement of cyclin A2 mRNA.
Dev Biol. 2010 Sep 15;345(2):156-69. doi: 10.1016/j.ydbio.2010.07.005. Epub 2010 Jul 13.

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