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[扩大膀胱成形术后膀胱延迟性自发性破裂]

[Delayed spontaneous rupture of the bladder following augmentation enterocystoplasty].

作者信息

Leibovitch I, Ramon J, Ben Chaim J, Goldwasser B

机构信息

Dept. of Urology, Chaim Sheba Medical Center, Tel Hashomer and Sackler School of Medicine, Tel Aviv University.

出版信息

Harefuah. 1990 Aug;119(3-4):68-70.

PMID:2227670
Abstract

Delayed spontaneous rupture of the urinary bladder following augmentation enterocystoplasty is a serious life-threatening complication of uncertain etiology. Multiple factors are believed to contribute to the mechanism of bladder perforation. Ruptured augmented bladders share a common urodynamic pattern of high leak point pressure of the urethra, with sensory and mechanical tolerance of high filling pressure. This combination seems to be the main predisposing factor for spontaneous perforation. Other risk factors, including catheter trauma during intermittent self-catheterization, urinary retention due to mucus retention or noncompliance with the catheterization protocol, chronic infection, and decreased sensation of bladder filling, may play roles in the mechanism of rupture. Clinically, patients present with sepsis, abdominal pain and distension, ileus, fever, oliguria and peritoneal irritation. The diagnosis is made on low pressure cystography, although failure of cystography to demonstrate extravasation is not unusual. Aggressive surgical treatment consists of immediate exploration, primary repair of the perforation, drainage of the perivesical space, suprapubic cystostomy and broad-spectrum antibiotics. Longterm management includes a strict intermittent catheterization schedule, anticholinergic therapy and urodynamic evaluation. Failure to achieve a low pressure storage reservoir by conservative means entails an increased risk of recurrent perforation. In such cases further surgical intervention should be considered. We present a 21-year-old paraplegic man 5 months after augmentation enterocystoplasty who required operation because of spontaneous rupture of the augmented bladder. Spontaneous delayed rupture of the bladder should be considered in the differential diagnosis of acute abdomen in patients after augmentation enterocystoplasty. Early surgical treatment and subsequent monitoring of the low pressure reservoir are recommended.

摘要

膀胱扩大肠囊成形术后膀胱延迟性自发性破裂是一种病因不明的严重危及生命的并发症。多种因素被认为与膀胱穿孔机制有关。破裂的扩大膀胱具有共同的尿动力学模式,即尿道高漏点压力,对高充盈压力具有感觉和机械耐受性。这种组合似乎是自发性穿孔的主要诱发因素。其他危险因素,包括间歇性自我导尿期间的导管创伤、由于黏液潴留或不遵守导尿方案导致的尿潴留、慢性感染以及膀胱充盈感觉减退,可能在破裂机制中起作用。临床上,患者表现为脓毒症、腹痛和腹胀、肠梗阻、发热、少尿和腹膜刺激征。诊断依靠低压膀胱造影,尽管膀胱造影未能显示外渗并不罕见。积极的手术治疗包括立即探查、穿孔的一期修复、膀胱周围间隙引流、耻骨上膀胱造瘘和广谱抗生素。长期管理包括严格的间歇性导尿计划、抗胆碱能治疗和尿动力学评估。通过保守方法未能实现低压储尿囊会增加复发性穿孔的风险。在这种情况下,应考虑进一步的手术干预。我们报告一名21岁截瘫男性,在膀胱扩大肠囊成形术后5个月因扩大膀胱的自发性破裂而需要手术。在膀胱扩大肠囊成形术后患者的急腹症鉴别诊断中应考虑膀胱自发性延迟破裂。建议早期手术治疗并随后监测低压储尿囊。

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