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Studies on Axenfeld-Rieger syndrome patients and mice reveal Foxc1's role in corneal neovascularization.对Axenfeld-Rieger综合征患者和小鼠的研究揭示了Foxc1在角膜新生血管形成中的作用。
Proc Natl Acad Sci U S A. 2012 Feb 7;109(6):1818-9. doi: 10.1073/pnas.1119291109. Epub 2012 Jan 30.
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Comparison of Bioinformatics Prediction, Molecular Modeling, and Functional Analyses of FOXC1 Mutations in Patients with Axenfeld-Rieger Syndrome.Axenfeld-Rieger综合征患者中FOXC1突变的生物信息学预测、分子建模及功能分析比较
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[Electron microscopic studies on the corneal neovascularization].[角膜新生血管形成的电子显微镜研究]
Nippon Ganka Gakkai Zasshi. 1969 Aug;73(8):1208-21.
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A Family with Axenfeld-Rieger Syndrome: Report of the Clinical and Genetic Findings.一个患有Axenfeld-Rieger综合征的家族：临床与遗传学发现报告
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Quantitation of neovascularization in flat preparations of the cornea.角膜平铺标本中新生血管形成的定量分析。
Microvasc Res. 1986 Jan;31(1):104-9. doi: 10.1016/0026-2862(86)90011-7.
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Argon laser treatment of corneal neovascularization.
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Axenfeld-Rieger syndrome in the age of molecular genetics.分子遗传学时代的Axenfeld-Rieger综合征
Am J Ophthalmol. 2000 Jul;130(1):107-15. doi: 10.1016/s0002-9394(00)00525-0.

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Diagnostic Challenges of Axenfeld-Rieger Syndrome and a Novel FOXC1 Gene Mutation in a Polish Family.波兰家族中Axenfeld-Rieger综合征的诊断挑战及一种新的FOXC1基因突变
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R-propranolol is a small molecule inhibitor of the SOX18 transcription factor in a rare vascular syndrome and hemangioma.R-普萘洛尔是一种罕见血管综合征和血管瘤中 SOX18 转录因子的小分子抑制剂。
Elife. 2019 Jul 30;8:e43026. doi: 10.7554/eLife.43026.
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FoxC1-dependent regulation of vascular endothelial growth factor signaling in corneal avascularity.FoxC1 依赖性调控角膜无血管区血管内皮生长因子信号通路
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本文引用的文献

1
Forkhead box transcription factor FoxC1 preserves corneal transparency by regulating vascular growth.叉头框转录因子 FoxC1 通过调节血管生长来保持角膜透明度。
Proc Natl Acad Sci U S A. 2012 Feb 7;109(6):2015-20. doi: 10.1073/pnas.1109540109. Epub 2011 Dec 14.
2
Primitive mesodermal cells with a neural crest stem cell phenotype predominate proliferating infantile haemangioma.具有神经嵴干细胞表型的原始中胚层细胞在增殖性婴儿血管瘤中占主导地位。
J Clin Pathol. 2010 Sep;63(9):771-6. doi: 10.1136/jcp.2010.079368. Epub 2010 Aug 9.
3
Specification of arterial, venous, and lymphatic endothelial cells during embryonic development.胚胎发育过程中动脉、静脉和淋巴管内皮细胞的特征。
Histol Histopathol. 2010 May;25(5):637-46. doi: 10.14670/HH-25.637.
4
FOXC1 is required for normal cerebellar development and is a major contributor to chromosome 6p25.3 Dandy-Walker malformation.FOXC1是正常小脑发育所必需的，并且是6号染色体p25.3区域丹迪-沃克畸形的主要成因。
Nat Genet. 2009 Sep;41(9):1037-42. doi: 10.1038/ng.422. Epub 2009 Aug 9.
5
FoxC1 is essential for vascular basement membrane integrity and hyaloid vessel morphogenesis.FoxC1对于血管基底膜完整性和玻璃体血管形态发生至关重要。
Invest Ophthalmol Vis Sci. 2009 Nov;50(11):5026-34. doi: 10.1167/iovs.09-3447. Epub 2009 May 20.
6
Combinatorial regulation of endothelial gene expression by ets and forkhead transcription factors.Ets和叉头转录因子对内皮基因表达的组合调控。
Cell. 2008 Dec 12;135(6):1053-64. doi: 10.1016/j.cell.2008.10.049.
7
Sox18 induces development of the lymphatic vasculature in mice.Sox18诱导小鼠淋巴管系统的发育。
Nature. 2008 Dec 4;456(7222):643-7. doi: 10.1038/nature07391. Epub 2008 Oct 19.
8
Corneal avascularity is due to soluble VEGF receptor-1.角膜无血管状态归因于可溶性血管内皮生长因子受体-1。
Nature. 2006 Oct 26;443(7114):993-7. doi: 10.1038/nature05249. Epub 2006 Oct 18.
9
Isolation of multipotent neural crest-derived stem cells from the adult mouse cornea.从成年小鼠角膜中分离多能神经嵴衍生干细胞。
Stem Cells. 2006 Dec;24(12):2714-22. doi: 10.1634/stemcells.2006-0156. Epub 2006 Aug 3.
10
The forkhead transcription factors, Foxc1 and Foxc2, are required for arterial specification and lymphatic sprouting during vascular development.叉头转录因子Foxc1和Foxc2是血管发育过程中动脉特化和淋巴管生成所必需的。
Dev Biol. 2006 Jun 15;294(2):458-70. doi: 10.1016/j.ydbio.2006.03.035. Epub 2006 Apr 3.

Studies on Axenfeld-Rieger syndrome patients and mice reveal Foxc1's role in corneal neovascularization.

作者信息

François Mathias, Ramchandran Ramani

机构信息

Institute for Molecular Bioscience, The University of Queensland, Brisbane, Queensland 4072, Australia.

出版信息

Proc Natl Acad Sci U S A. 2012 Feb 7;109(6):1818-9. doi: 10.1073/pnas.1119291109. Epub 2012 Jan 30.

DOI:10.1073/pnas.1119291109
PMID:22308435
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3277551/
Abstract
摘要