Sismanis A, Callari R H, Slomka W S, Butts F M
Department of Otolaryngology, Medical College of Virginia, Virginia Commonwealth University, Richmond.
Laryngoscope. 1990 Nov;100(11):1152-5. doi: 10.1288/00005537-199011000-00003.
In this study, auditory brainstem-evoked responses were conducted on 28 patients with otologic symptoms (pulsatile tinnitus, hearing loss, aural fullness) secondary to benign intracranial hypertension syndrome. Abnormalities consisting mainly of prolonged interpeak latencies were detected in one third of these patients. It is speculated that the pathophysiologic mechanisms responsible for these auditory brainstem-evoked abnormalities are stretching-compression of the cochlear nerve and brainstem caused by the intracranial hypertension and/or primary edema of the same structures due to the benign intracranial hypertension syndrome itself. Normalization or improvement was noticed in the majority of the patients after management. Since the number of patients in this study is small, it is felt that the diagnostic and prognostic value of this test needs further evaluation.
在本研究中,对28例继发于良性颅内高压综合征且有耳科症状(搏动性耳鸣、听力损失、耳闷)的患者进行了听觉脑干诱发电位检测。在这些患者中,三分之一检测到主要由峰间期延长组成的异常情况。据推测,导致这些听觉脑干诱发异常的病理生理机制是颅内高压引起的蜗神经和脑干的拉伸-压迫,和/或由于良性颅内高压综合征本身导致的相同结构的原发性水肿。在进行相应处理后,大多数患者的情况恢复正常或有所改善。由于本研究中的患者数量较少,认为该检查的诊断和预后价值需要进一步评估。