Haller K A, Miller-Meeks M, Kardon R
Department of Ophthalmology, University of Iowa Hospitals and Clinics, Iowa City 52242.
Ophthalmology. 1990 Sep;97(9):1162-5. doi: 10.1016/s0161-6420(90)32441-7.
Adduction deficiency following acute head trauma may result not only from orbital damage but also from internuclear ophthalmoplegia, and in most instances this resolves over weeks to months. To date, noninvasive imaging studies during the acute phase following injury have not been definitive in localizing the pathology. Three cases of adduction deficiency following head trauma that were caused by internuclear ophthalmoplegia are reported. A lesion in the brain stem was found in all three cases by magnetic resonance imaging in the subacute post-traumatic period. These lesions were not visible on routine x-ray computed tomography obtained at the time of injury.
急性头部创伤后内收功能障碍不仅可能由眼眶损伤引起,也可能由核间性眼肌麻痹导致,在大多数情况下,这种情况会在数周数月内自行缓解。迄今为止,损伤急性期的非侵入性影像学检查在确定病变位置方面尚未明确。本文报告了3例因核间性眼肌麻痹导致头部创伤后内收功能障碍的病例。在创伤后亚急性期,通过磁共振成像在所有3例病例中均发现脑干有病变。这些病变在受伤时进行的常规X线计算机断层扫描中不可见。