Selvaraj Raja J, Gobu Pakkirisamy, Ashida Thulaseedharan S, George Geofi, Balachander Jayaraman
Department of Cardiology, Jawaharlal Institute of Postgraduate Medical Education and Research.
Indian Pacing Electrophysiol J. 2012 Jan;12(1):27-31. doi: 10.1016/s0972-6292(16)30462-4. Epub 2012 Jan 31.
A 35 year old female presented with recurrent ventricular tachycardia 5 years after she had undergone surgical repair of double chambered right ventricle. Electroanatomical mapping showed a localised scar in the apex with double potentials and good pace map. Ablation here resulted in non-inducibility of ventricular tachycardia. We hypothesise that the scarring in the apex is the result of sustained pressure overload and becomes arrhythmogenic similar to the apical scar in patients with mid-ventricular hypertrophic cardiomyopathy.
一名35岁女性在接受双腔右心室手术修复5年后出现反复发作的室性心动过速。电解剖标测显示心尖部有局限性瘢痕,伴有双电位且起搏标测良好。在此处进行消融导致室性心动过速不能被诱发。我们推测心尖部的瘢痕是持续压力负荷过重的结果,并且变得具有致心律失常性,类似于心室中部肥厚型心肌病患者的心尖部瘢痕。