Decavel Pierre, Vuillier Fabrice, Moulin Thierry
Service de Neurologie, CHU Besançon, Besançon, France.
Front Neurol Neurosci. 2012;30:115-9. doi: 10.1159/000333606. Epub 2012 Feb 14.
Lenticulostriate infarcts result from ischemia within the territory supplied by the deep perforating branches of the middle cerebral artery (MCA). They are too often associated with infarctions of the deep perforating branches of the internal carotid artery. Lenticulostriate arteries usually arise from the main trunk of the MCA, but can emerge from the cortical branches. The clinical aspects of lenticulostriate infarction should be properly differentiated from those of other anterior circulation infarcts. Clinical signs include motor deficit, sensory deficit and cognitive dysfunction. The principal mechanism for lenticulostriate infarction seems to be an embolism of cardiac origin. The concept of lacunar infarctions relating to lipohyalinosis is perhaps too often proposed without evidence. The prognosis is dependent primarily on the intensity of damage to the upper part of the posterior limb of the internal capsule. They are terminal arteries without anastomoses, making them more susceptible to ischemia and resulting in a greater risk of arteriolar necrosis and hemorrhagic transformation.
豆纹状梗死是由大脑中动脉（MCA）深穿支供血区域内的缺血引起的。它们常常与颈内动脉深穿支梗死相关。豆纹状动脉通常起源于MCA主干，但也可发自皮质支。豆纹状梗死的临床特征应与其他前循环梗死的特征进行恰当区分。临床体征包括运动功能缺损、感觉功能缺损和认知功能障碍。豆纹状梗死的主要机制似乎是心源性栓塞。与脂质透明变性相关的腔隙性梗死概念常常在没有证据的情况下被提出。预后主要取决于内囊后肢上部的损伤程度。它们是没有吻合支的终末动脉，这使得它们更容易发生缺血，并导致小动脉坏死和出血性转化的风险更高。
