吡那地尔和钙对离体大鼠心脏线粒体的影响。 - Suppr

Effects of pinacidil and calcium on isolated rat heart mitochondria.

作者信息

Korotkov S M, Emel'yanova L V, Brailovskaya I V, Nesterov V P

机构信息

Sechenov Institute of Evolutionary Physiology and Biochemistry, Russian Academy of Sciences, pr. Morisa Toreza 44, St. Petersburg, 194223 Russia.

出版信息

Dokl Biochem Biophys. 2012 Mar-Apr;443:113-7. doi: 10.1134/S1607672912020147. Epub 2012 May 5.

DOI:10.1134/S1607672912020147

PMID:22562638

Abstract

摘要

相似文献

Effects of pinacidil and calcium on isolated rat heart mitochondria.吡那地尔和钙对离体大鼠心脏线粒体的影响。

Dokl Biochem Biophys. 2012 Mar-Apr;443:113-7. doi: 10.1134/S1607672912020147. Epub 2012 May 5.

Effects of Pinacidil and Calcium on Succinate-Energized Rat Heart Mitochondria in the Presence of Rotenone.在存在鱼藤酮的情况下，匹那地尔和钙对琥珀酸供能的大鼠心脏线粒体的影响。

Dokl Biochem Biophys. 2019 Jul;487(1):277-281. doi: 10.1134/S1607672919040070. Epub 2019 Sep 26.

Mersalyl prevents the Tl-induced permeability transition pore opening in the inner membrane of Ca-loaded rat liver mitochondria.汞撒利可防止铊诱导的钙负载大鼠肝线粒体内膜通透性转换孔开放。

Biochem Biophys Res Commun. 2018 Jan 8;495(2):1716-1721. doi: 10.1016/j.bbrc.2017.12.023. Epub 2017 Dec 6.

Closure of mitochondrial potassium channels favors opening of the Tl(+)-induced permeability transition pore in Ca(2+)-loaded rat liver mitochondria.线粒体钾通道的关闭有利于在钙离子负载的大鼠肝脏线粒体中，铊离子诱导的通透性转换孔的开放。

J Bioenerg Biomembr. 2015 Jun;47(3):243-54. doi: 10.1007/s10863-015-9611-2. Epub 2015 Apr 14.

Deregulation of mitochondrial functions provoked by long-chain fatty acid accumulating in long-chain 3-hydroxyacyl-CoA dehydrogenase and mitochondrial permeability transition deficiencies in rat heart--mitochondrial permeability transition pore opening as a potential contributing pathomechanism of cardiac alterations in these disorders.长链脂肪酸在大鼠心脏中积累引发的线粒体功能失调，与长链3-羟酰基辅酶A脱氢酶及线粒体通透性转换缺陷有关——线粒体通透性转换孔开放是这些疾病中心脏改变的潜在病理机制。

FEBS J. 2015 Dec;282(24):4714-26. doi: 10.1111/febs.13526. Epub 2015 Oct 12.

[Hydrogen sulfide inhibits Ca(2+)-induced mitochondrial permeability transition pore opening in spontaneously hypertensive rats].[硫化氢抑制自发性高血压大鼠中钙离子诱导的线粒体通透性转换孔开放]

Fiziol Zh (1994). 2013;59(1):3-10.

Tl(+) induces both cationic and transition pore permeability in the inner membrane of rat heart mitochondria.Tl(+) 诱导大鼠心肌线粒体内膜的阳离子和过渡孔通透性。

J Bioenerg Biomembr. 2013 Dec;45(6):531-9. doi: 10.1007/s10863-013-9526-8. Epub 2013 Sep 22.

[Hydrogen sulfide inhibits Ca(2+)-induced mitochondrial permeability transition pore opening in adult and old rat heart].硫化氢抑制成年及老年大鼠心脏中钙离子诱导的线粒体通透性转换孔开放

Fiziol Zh (1994). 2011;57(6):3-14.

Silencing of cardiac mitochondrial NHE1 prevents mitochondrial permeability transition pore opening.沉默心脏线粒体中的 NHE1 可防止线粒体通透性转换孔开放。

Am J Physiol Heart Circ Physiol. 2011 Apr;300(4):H1237-51. doi: 10.1152/ajpheart.00840.2010. Epub 2011 Feb 4.

Hexokinase II dissociation alone cannot account for changes in heart mitochondrial function, morphology and sensitivity to permeability transition pore opening following ischemia.单纯的己糖激酶 II 分离不能解释缺血后心脏线粒体功能、形态和对通透性转换孔开放敏感性的变化。

PLoS One. 2020 Jun 24;15(6):e0234653. doi: 10.1371/journal.pone.0234653. eCollection 2020.

引用本文的文献

Dokl Biochem Biophys. 2019 Jul;487(1):277-281. doi: 10.1134/S1607672919040070. Epub 2019 Sep 26.

J Bioenerg Biomembr. 2015 Jun;47(3):243-54. doi: 10.1007/s10863-015-9611-2. Epub 2015 Apr 14.

Tl(+) induces both cationic and transition pore permeability in the inner membrane of rat heart mitochondria.Tl(+) 诱导大鼠心肌线粒体内膜的阳离子和过渡孔通透性。

J Bioenerg Biomembr. 2013 Dec;45(6):531-9. doi: 10.1007/s10863-013-9526-8. Epub 2013 Sep 22.

本文引用的文献

Direct effects of K(ATP) channel openers pinacidil and diazoxide on oxidative phosphorylation of mitochondria in situ.钾离子通道开放剂吡那地尔和二氮嗪对原位线粒体氧化磷酸化的直接作用。

Cell Physiol Biochem. 2010;25(2-3):181-6. doi: 10.1159/000276552. Epub 2010 Jan 12.

Interaction of mitochondrial potassium channels with the permeability transition pore.线粒体钾通道与通透性转换孔的相互作用。

FEBS Lett. 2010 May 17;584(10):2005-12. doi: 10.1016/j.febslet.2009.12.038. Epub 2009 Dec 27.

KATP channel openers have opposite effects on mitochondrial respiration under different energetic conditions.在不同能量条件下，ATP敏感性钾通道开放剂对线粒体呼吸具有相反的作用。

J Cardiovasc Pharmacol. 2008 May;51(5):483-91. doi: 10.1097/FJC.0b013e31816bf4a4.

The direct physiological effects of mitoK(ATP) opening on heart mitochondria.线粒体ATP敏感性钾通道开放对心脏线粒体的直接生理效应。

Am J Physiol Heart Circ Physiol. 2006 Jan;290(1):H406-15. doi: 10.1152/ajpheart.00794.2005. Epub 2005 Sep 2.

Mitochondrial K(ATP) channels in cell survival and death.细胞存活与死亡中的线粒体ATP敏感性钾通道

J Mol Cell Cardiol. 2005 Jul;39(1):7-16. doi: 10.1016/j.yjmcc.2004.12.003. Epub 2005 Feb 19.

Potassium channel openers are uncoupling protonophores: implication in cardioprotection.钾通道开放剂是解偶联质子载体：对心脏保护的意义。

FEBS Lett. 2004 Jun 18;568(1-3):167-70. doi: 10.1016/j.febslet.2004.05.031.

Preconditioning protects by inhibiting the mitochondrial permeability transition.预处理通过抑制线粒体通透性转换发挥保护作用。

Am J Physiol Heart Circ Physiol. 2004 Aug;287(2):H841-9. doi: 10.1152/ajpheart.00678.2003. Epub 2004 Apr 8.

The adenine nucleotide translocase: a central component of the mitochondrial permeability transition pore and key player in cell death.腺嘌呤核苷酸转位酶：线粒体通透性转换孔的核心组成部分及细胞死亡的关键参与者。

Curr Med Chem. 2003 Aug;10(16):1507-25. doi: 10.2174/0929867033457278.

K-ATP channel independent effects of pinacidil on ATP production in isolated cardiomyocyte or pancreatic beta-cell mitochondria.吡那地尔对分离的心肌细胞或胰岛β细胞线粒体中ATP生成的K-ATP通道非依赖性作用。

Biochem Pharmacol. 2003 Jun 1;65(11):1835-41. doi: 10.1016/s0006-2952(03)00179-5.

K(ATP) channel-independent targets of diazoxide and 5-hydroxydecanoate in the heart.心脏中地尔硫䓬和5-羟基癸酸酯不依赖于K(ATP)通道的靶点。

J Physiol. 2002 Aug 1;542(Pt 3):735-41. doi: 10.1113/jphysiol.2002.023960.

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Effects of pinacidil and calcium on isolated rat heart mitochondria.

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