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Distribution of cardiac glycosides in heart and brain of dogs and their affinity to the (Na+ + K+)-ATPase.

作者信息

Kuhlmann J, Erdmann E, Rietbrock N

出版信息

Naunyn Schmiedebergs Arch Pharmacol. 1979 May;307(1):65-71. doi: 10.1007/BF00506553.

DOI:10.1007/BF00506553
PMID:225683
Abstract
摘要

相似文献

1
Distribution of cardiac glycosides in heart and brain of dogs and their affinity to the (Na+ + K+)-ATPase.强心苷在犬心脏和大脑中的分布及其对(钠+钾)-ATP酶的亲和力。
Naunyn Schmiedebergs Arch Pharmacol. 1979 May;307(1):65-71. doi: 10.1007/BF00506553.
2
A kinetic comparison of cardiac glycoside interactions with Na+,K+-ATPases from skeletal and cardiac muscle and from kidney.
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3
Isoform specificity of cardiac glycosides binding to human Na+,K+-ATPase alpha1beta1, alpha2beta1 and alpha3beta1.心脏糖苷与人 Na+,K+-ATPase α1β1、α2β1 和 α3β1 结合的异构体特异性。
Eur J Pharmacol. 2009 Nov 10;622(1-3):7-14. doi: 10.1016/j.ejphar.2009.08.039. Epub 2009 Sep 12.
4
Interaction of cardiac glycosides with Na+,K+-ATPase.强心苷与钠钾ATP酶的相互作用。
Methods Enzymol. 1988;156:201-13. doi: 10.1016/0076-6879(88)56022-6.
5
Cryptosin induces backbone structural changes in cardiac Na+ and K+ dependent adenosinetriphosphatase.隐丹参酮诱导心脏钠钾依赖三磷酸腺苷酶的主链结构变化。
Biochem Int. 1990 Oct;22(2):287-94.
6
Reduction of the equilibrium binding of cardiac glycosides and related compounds to Na+,K+-ATPase as a possible mechanism for the potassium-induced reversal of their toxicity.强心苷及相关化合物与钠钾ATP酶平衡结合的减少,可能是钾诱导其毒性逆转的一种机制。
Naunyn Schmiedebergs Arch Pharmacol. 1978 Sep;304(2):157-65. doi: 10.1007/BF00495552.
7
Interaction of digoxin and SC4453 with the (Na+ + K+)-ATPase from guinea-pig heart and brain.地高辛和SC4453与豚鼠心脏和大脑的(钠+钾)-ATP酶的相互作用。
Arch Int Pharmacodyn Ther. 1979 May;239(1):171-2.
8
Retinoschisin and Cardiac Glycoside Crosstalk at the Retinal Na/K-ATPase.视蛋白聚糖与心脏糖苷在视网膜 Na/K-ATP 酶上的相互作用。
Invest Ophthalmol Vis Sci. 2020 May 11;61(5):1. doi: 10.1167/iovs.61.5.1.
9
Saturable adenosine 5'-triphosphate-independent binding of [3H]-ouabain to brain and cardiac tissue in vitro.[3H]哇巴因在体外与脑和心脏组织的可饱和的、不依赖三磷酸腺苷的结合
Br J Pharmacol. 1979 Mar;65(3):403-9. doi: 10.1111/j.1476-5381.1979.tb07844.x.
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The interaction of ouabagenin and (Na+ + K+)-ATPase in the presence of Na+, Mg2+, and ATP.
Mol Pharmacol. 1981 Jan;19(1):62-7.

引用本文的文献

1
Identification of a Cardiac Glycoside Exhibiting Favorable Brain Bioavailability and Potency for Reducing Levels of the Cellular Prion Protein.鉴定一种具有良好脑生物利用度和降低细胞朊病毒蛋白水平效力的心脏糖苷。
Int J Mol Sci. 2022 Nov 26;23(23):14823. doi: 10.3390/ijms232314823.
2
Cardiac glycoside-mediated turnover of Na, K-ATPases as a rational approach to reducing cell surface levels of the cellular prion protein.心脏糖苷介导的 Na,K-ATP 酶周转作为降低细胞表面细胞朊病毒蛋白水平的合理方法。
PLoS One. 2022 Jul 1;17(7):e0270915. doi: 10.1371/journal.pone.0270915. eCollection 2022.

本文引用的文献

1
Protein measurement with the Folin phenol reagent.使用福林酚试剂进行蛋白质测定。
J Biol Chem. 1951 Nov;193(1):265-75.
2
THE ACTION OF CARDIAC GLYCOSIDES ON ION MOVEMENTS.强心苷对离子运动的作用。
Pharmacol Rev. 1964 Dec;16:381-407.
3
[ON THE IDENTITY OF THE ION-PUMPING-ATPASE IN THE CELL MEMBRANE OF THE MYOCARDIUM WITH A DIGITALIS RECEPTOR ENZYME].[关于心肌细胞膜中离子泵ATP酶与洋地黄受体酶的同一性]
Experientia. 1963 Sep 15;19:452-8. doi: 10.1007/BF02150643.
4
Studies on sodium-potassium-activated adenosine triphosphatase. I. Quantitative distribution in several tissues of the cat.钠钾激活三磷酸腺苷酶的研究。I. 猫几种组织中的定量分布。
Arch Biochem Biophys. 1961 Dec;95:416-23. doi: 10.1016/0003-9861(61)90170-9.
5
The absence of blood-brain barrier from certain areas of the central nervous system.
J Pharmacol Exp Ther. 1961 Sep;133:332-4.
6
Demonstration of an ATPase at the cell surface of intact normal and neoplastic human cells in culture.完整的正常和肿瘤人类培养细胞表面ATP酶的展示。
J Cell Physiol. 1971 Oct;78(2):171-6. doi: 10.1002/jcp.1040780203.
7
ATPase in human brain tumors.人类脑肿瘤中的ATP酶
J Neurosurg. 1970 Aug;33(2):167-71. doi: 10.3171/jns.1970.33.2.0167.
8
[Properties of the receptor for cardiac glycosides (author's transl)].[强心苷受体的特性(作者译)]
Klin Wochenschr. 1974 Aug 1;52(15):705-18. doi: 10.1007/BF01469333.
9
Ouabain-receptor interactions in (Na + +K + )-ATPase preparations from different tissues and species. Determination of kinetic constants and dissociation constants.来自不同组织和物种的(钠钾)-ATP酶制剂中哇巴因与受体的相互作用。动力学常数和解离常数的测定。
Biochim Biophys Acta. 1973 May 11;307(2):386-98. doi: 10.1016/0005-2736(73)90104-1.
10
Correlation of cardiac sodium- and potassium-activated adenosine triphosphatase activity with ouabain-induced inotropic stimulation.心脏钠钾激活三磷酸腺苷酶活性与哇巴因诱导的变力性刺激的相关性
J Pharmacol Exp Ther. 1970 May;173(1):145-51.