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Expression of the autoimmunity associated TNFAIP3 is increased in rheumatoid arthritis but does not differ according to genotype at 6q23.

作者信息

Maxwell James R, Gowers Isobel R, Kuet Kar-Ping, Barton Anne, Worthington Jane, Wilson Anthony G

出版信息

Rheumatology (Oxford). 2012 Aug;51(8):1514-5. doi: 10.1093/rheumatology/kes134. Epub 2012 May 22.

DOI:10.1093/rheumatology/kes134
PMID:22623711
Abstract
摘要

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Expression of the autoimmunity associated TNFAIP3 is increased in rheumatoid arthritis but does not differ according to genotype at 6q23.自身免疫相关的TNFAIP3在类风湿性关节炎中的表达增加，但在6q23位点上其表达根据基因型并无差异。
Rheumatology (Oxford). 2012 Aug;51(8):1514-5. doi: 10.1093/rheumatology/kes134. Epub 2012 May 22.
2
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3
Analysis of TNFAIP3, a feedback inhibitor of nuclear factor-kappaB and the neighbor intergenic 6q23 region in rheumatoid arthritis susceptibility.类风湿关节炎易感性中核因子-κB的反馈抑制剂TNFAIP3及相邻基因间6q23区域的分析。
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Studying associations between variants in TRAF1-C5 and TNFAIP3-OLIG3 and the progression of joint destruction in rheumatoid arthritis in multiple cohorts.在多个队列中研究TRAF1-C5和TNFAIP3-OLIG3基因变异与类风湿关节炎关节破坏进展之间的关联。
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5
Complex genetic association of 6q23 with autoimmune rheumatic conditions.6q23与自身免疫性风湿性疾病的复杂基因关联。
Arthritis Res Ther. 2009;11(2):107. doi: 10.1186/ar2663. Epub 2009 Apr 27.
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Genetic variants near TNFAIP3 on 6q23 are associated with systemic lupus erythematosus.位于6号染色体23区带的TNFAIP3基因附近的遗传变异与系统性红斑狼疮相关。
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A Tunisian case-control association study of a 6q polymorphism in rheumatoid arthritis.一项关于类风湿关节炎 6q 多态性的突尼斯病例对照关联研究。
Rheumatol Int. 2012 Jun;32(6):1849-50. doi: 10.1007/s00296-011-1996-6. Epub 2011 Jul 22.
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Brief report: candidate gene study in systemic sclerosis identifies a rare and functional variant of the TNFAIP3 locus as a risk factor for polyautoimmunity.简短报告：系统性硬化症的候选基因研究确定TNFAIP3基因座的一种罕见功能性变异是多自身免疫的危险因素。
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TNFAIP3 rs2230926 polymorphisms in rheumatoid arthritis of southern Chinese Han population: a case-control study.中国南方汉族人群类风湿关节炎中TNFAIP3基因rs2230926多态性：一项病例对照研究。
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Combined effects of three independent SNPs greatly increase the risk estimate for RA at 6q23.三个独立单核苷酸多态性的联合效应极大地增加了6号染色体长臂23区类风湿性关节炎的风险评估。
Hum Mol Genet. 2009 Jul 15;18(14):2693-9. doi: 10.1093/hmg/ddp193. Epub 2009 May 5.

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T Cell Transcriptomes from Paroxysmal Nocturnal Hemoglobinuria Patients Reveal Novel Signaling Pathways.阵发性睡眠性血红蛋白尿症患者的T细胞转录组揭示了新的信号通路。
J Immunol. 2017 Jul 15;199(2):477-488. doi: 10.4049/jimmunol.1601299. Epub 2017 Jun 19.
2
Three single nucleotide polymorphisms of TNFAIP3 gene increase the risk of rheumatoid arthritis.TNFAIP3基因的三个单核苷酸多态性增加类风湿性关节炎的风险。
Oncotarget. 2017 Mar 28;8(13):20784-20793. doi: 10.18632/oncotarget.15265.
3
TNFAIP3 gene rs10499194, rs13207033 polymorphisms decrease the risk of rheumatoid arthritis.
TNFAIP3基因rs10499194、rs13207033多态性降低类风湿关节炎风险。
Oncotarget. 2016 Dec 13;7(50):82933-82942. doi: 10.18632/oncotarget.12638.
4
Altered mRNA expression in peripheral blood mononuclear cells from patients with rheumatoid arthritis.
Biomed Rep. 2015 Sep;3(5):675-680. doi: 10.3892/br.2015.486. Epub 2015 Jun 30.
5
Specific overexpression of tumour necrosis factor-α-induced protein (TNFAIP)9 in CD14(+) CD16(-) monocytes in patients with rheumatoid arthritis: comparative analysis with TNFAIP3.类风湿关节炎患者CD14(+) CD16(-) 单核细胞中肿瘤坏死因子-α诱导蛋白（TNFAIP）9的特异性过表达：与TNFAIP3的比较分析
Clin Exp Immunol. 2015 Jun;180(3):458-66. doi: 10.1111/cei.12606.
6
Measles contributes to rheumatoid arthritis: evidence from pathway and network analyses of genome-wide association studies.麻疹可导致类风湿性关节炎：基于全基因组关联研究的途径和网络分析的证据。
PLoS One. 2013 Oct 18;8(10):e75951. doi: 10.1371/journal.pone.0075951. eCollection 2013.
7
Influence of vitamin D status and vitamin D3 supplementation on genome wide expression of white blood cells: a randomized double-blind clinical trial.维生素 D 状态和维生素 D3 补充对白细胞全基因组表达的影响：一项随机、双盲临床试验。
PLoS One. 2013;8(3):e58725. doi: 10.1371/journal.pone.0058725. Epub 2013 Mar 20.