Forever young: induced pluripotent stem cells as models of inherited arrhythmias.
作者信息
Park David S, Fishman Glenn I
出版信息
Circulation. 2012 Jun 26;125(25):3055-6. doi: 10.1161/CIRCULATIONAHA.112.114165. Epub 2012 May 30.
Abstract
摘要
相似文献
1
Forever young: induced pluripotent stem cells as models of inherited arrhythmias.永葆青春：诱导多能干细胞作为遗传性心律失常的模型
Circulation. 2012 Jun 26;125(25):3055-6. doi: 10.1161/CIRCULATIONAHA.112.114165. Epub 2012 May 30.
2
Cardiomyocytes derived from pluripotent stem cells recapitulate electrophysiological characteristics of an overlap syndrome of cardiac sodium channel disease.多能干细胞衍生的心肌细胞再现了心脏钠离子通道病重叠综合征的电生理特征。
Circulation. 2012 Jun 26;125(25):3079-91. doi: 10.1161/CIRCULATIONAHA.111.066092. Epub 2012 May 30.
3
Modeling genetic cardiac channelopathies using induced pluripotent stem cells - Status quo from an electrophysiological perspective.使用诱导多能干细胞对遗传性心脏通道病进行建模——从电生理学角度看现状。
Biochem Pharmacol. 2021 Oct;192:114746. doi: 10.1016/j.bcp.2021.114746. Epub 2021 Aug 27.
4
Concise Review: Cardiac Disease Modeling Using Induced Pluripotent Stem Cells.简要综述：利用诱导多能干细胞进行心脏疾病建模
Stem Cells. 2015 Sep;33(9):2643-51. doi: 10.1002/stem.2070. Epub 2015 Jun 23.
5
Human Cardiac Organoids for Disease Modeling.人类心脏类器官用于疾病建模。
Clin Pharmacol Ther. 2019 Jan;105(1):79-85. doi: 10.1002/cpt.1286. Epub 2018 Dec 21.
6
A consistent arrhythmogenic trait in Brugada syndrome cellular phenotype.Brugada综合征细胞表型中一种一致的致心律失常特征。
Clin Transl Med. 2021 Jun;11(6):e413. doi: 10.1002/ctm2.413.
7
Cardiac Kir2.1 and Na1.5 Channels Traffic Together to the Sarcolemma to Control Excitability.心脏 Kir2.1 和 Na1.5 通道共同转运至肌浆网以控制兴奋性。
Circ Res. 2018 May 25;122(11):1501-1516. doi: 10.1161/CIRCRESAHA.117.311872. Epub 2018 Mar 7.
8
Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes: The New Working Horse in Cardiovascular Pharmacology?人诱导多能干细胞衍生的心肌细胞：心血管药理学中的新主力？
J Cardiovasc Pharmacol. 2021 Mar 1;77(3):265-266. doi: 10.1097/FJC.0000000000000981.
9
Safe and Effective Cardiac Regenerative Therapy With Human-Induced Pluripotent Stem Cells: How Should We Prepare Pure Cardiac Myocytes?用人诱导多能干细胞进行安全有效的心脏再生治疗：我们应如何制备纯心肌细胞？
Circ Res. 2017 May 12;120(10):1558-1560. doi: 10.1161/CIRCRESAHA.116.310328.
10
Cardiomyocytes obtained from induced pluripotent stem cells with long-QT syndrome 3 recapitulate typical disease-specific features in vitro.诱导多能干细胞来源的长 QT 综合征 3 型心肌细胞在体外再现典型的疾病特异性特征。
Circ Res. 2011 Sep 30;109(8):841-7. doi: 10.1161/CIRCRESAHA.111.243139. Epub 2011 Jul 28.
本文引用的文献
1
Cardiomyocytes derived from pluripotent stem cells recapitulate electrophysiological characteristics of an overlap syndrome of cardiac sodium channel disease.多能干细胞衍生的心肌细胞再现了心脏钠离子通道病重叠综合征的电生理特征。
Circulation. 2012 Jun 26;125(25):3079-91. doi: 10.1161/CIRCULATIONAHA.111.066092. Epub 2012 May 30.
2
Striking In vivo phenotype of a disease-associated human SCN5A mutation producing minimal changes in vitro.在体表现出与疾病相关的人类 SCN5A 突变的显著表型，而在体外仅有微小变化。
Circulation. 2011 Aug 30;124(9):1001-11. doi: 10.1161/CIRCULATIONAHA.110.987248. Epub 2011 Aug 8.
3
Modelling the long QT syndrome with induced pluripotent stem cells.利用诱导多能干细胞构建长 QT 综合征模型。
Nature. 2011 Mar 10;471(7337):225-9. doi: 10.1038/nature09747. Epub 2011 Jan 16.
4
Patient-specific induced pluripotent stem-cell models for long-QT syndrome.长 QT 综合征的患者特异性诱导多能干细胞模型。
N Engl J Med. 2010 Oct 7;363(15):1397-409. doi: 10.1056/NEJMoa0908679. Epub 2010 Jul 21.
5
Properties of WT and mutant hERG K(+) channels expressed in neonatal mouse cardiomyocytes.WT 和突变型 hERG K(+) 通道在新生鼠心肌细胞中的特性。
Am J Physiol Heart Circ Physiol. 2010 Jun;298(6):H1842-9. doi: 10.1152/ajpheart.01236.2009. Epub 2010 Apr 2.
6
The action potential and comparative pharmacology of stem cell-derived human cardiomyocytes.干细胞衍生的人类心肌细胞的动作电位及比较药理学
J Pharmacol Toxicol Methods. 2010 May-Jun;61(3):277-86. doi: 10.1016/j.vascn.2010.01.014. Epub 2010 Feb 11.
7
Functional characterization of cardiomyocytes derived from murine induced pluripotent stem cells in vitro.在体外从鼠诱导多能干细胞中分离的心肌细胞的功能特征。
FASEB J. 2009 Dec;23(12):4168-80. doi: 10.1096/fj.08-128546. Epub 2009 Aug 24.
8
Sodium channel mutations and arrhythmias.钠通道突变与心律失常。
Nat Rev Cardiol. 2009 May;6(5):337-48. doi: 10.1038/nrcardio.2009.44.
9
Readthrough of nonsense mutation W822X in the SCN5A gene can effectively restore expression of cardiac Na+ channels.SCN5A基因无义突变W822X的通读可有效恢复心脏钠离子通道的表达。
Cardiovasc Res. 2009 Aug 1;83(3):473-80. doi: 10.1093/cvr/cvp116. Epub 2009 Apr 17.
10
Gating properties of SCN5A mutations and the response to mexiletine in long-QT syndrome type 3 patients.长QT综合征3型患者中SCN5A突变的门控特性及对美西律的反应
Circulation. 2007 Sep 4;116(10):1137-44. doi: 10.1161/CIRCULATIONAHA.107.707877. Epub 2007 Aug 13.
Zotero 插件