Minamiguchi Hitoshi, Mizuno Hiroya, Masuda Masaharu, Sakata Yasushi, Saito Shunsuke, Nanto Shinsuke, Sawa Yoshiki, Komuro Issei
Department of Cardiovascular Medicine, Osaka University Graduate School of Medicine, Japan.
Int Heart J. 2012;53(2):146-8. doi: 10.1536/ihj.53.146.
A 19-year-old man who underwent bicaval orthotopic heart transplantation for idiopathic dilated cardiomyopathy complained of palpitations 2 weeks after the heart transplantation. An ECG revealed paroxysmal atrial tachycardia (AT) with a cycle length of 260 ms and the P wave morphology of the AT was similar to that during sinus rhythm. Echocardiography showed normal contraction. No rejection, vasculopathy, or infection was observed. An electrophysiologic study and catheter ablation guided by a noncontact mapping system were performed due to drug refractory AT. The AT was induced spontaneously by isoproterenol infusion. The activation sequence of the AT exhibited a focal pattern, and the breakout site of the AT into the donor right atrium was just 12 mm below the breakout site of the donor sinoatrial node. Radiofrequency catheter ablation eliminated this AT and resulted in an improvement in the symptoms.
一名19岁男性因特发性扩张型心肌病接受双腔原位心脏移植,心脏移植术后2周出现心悸。心电图显示阵发性房性心动过速(AT),周期长度为260毫秒,AT的P波形态与窦性心律时相似。超声心动图显示收缩正常。未观察到排斥反应、血管病变或感染。由于药物难治性AT,进行了非接触标测系统引导下的电生理研究和导管消融。静脉注射异丙肾上腺素可自发诱发AT。AT的激动顺序呈局灶性模式,AT进入供体右心房的突破点恰好在供体窦房结突破点下方12毫米处。射频导管消融消除了该AT,症状得到改善。