Bennett D, Artzt K
Department of Zoology, University of Texas, Austin 78712-1064.
Genet Res. 1990 Oct-Dec;56(2-3):179-83. doi: 10.1017/s0016672300035278.
We present data on the effects of three chromosome 17 deletions on transmission ratio distortion (TRD) and sterility of several t-haplotypes. All three deletions have similar effects on male TRD: that is, Tdel/tcomplete genotypes all transmit their t-haplotype in very high proportion. However, each deletion has different effects on sterility of heterozygous males, with TOr/t being fertile, Thp/t less fertile, and TOrl/t still less fertile. These data suggest that wild-type genes on chromosomes homologous to t-haplotypes can be important regulators of both TRD and fertility in males, and that the wild-type genes concerned with TRD and fertility are at least to some extent different. The data also provide a rough map of the positions of these genes.
我们展示了关于17号染色体上三个缺失对几种t单倍型的传递比率畸变（TRD）和不育性影响的数据。所有这三个缺失对雄性TRD具有相似的影响：也就是说，Tdel/tcomplete基因型都以非常高的比例传递它们的t单倍型。然而，每个缺失对杂合雄性的不育性有不同的影响，其中TOr/t可育，Thp/t育性较低，而TOrl/t育性更低。这些数据表明，与t单倍型同源的染色体上的野生型基因可能是雄性TRD和育性的重要调节因子，并且与TRD和育性相关的野生型基因至少在一定程度上是不同的。这些数据还提供了这些基因位置的大致图谱。
