Retinoids, vitamin D, invasion, and metastasis.
作者信息
Tokar E J, Ablin R J, Webber M M
机构信息
Inorganic Carcinogenesis Section, National Cancer Institute at the National Institute of Environmental Health Sciences, Research Triangle Park, North Carolina, U.S.A.
出版信息
Curr Oncol. 2006 Dec;13(6):198-200. doi: 10.3747/co.v13i6.120.
Abstract
摘要
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本文引用的文献
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Stem/progenitor and intermediate cell types and the origin of human prostate cancer.干细胞/祖细胞及中间细胞类型与人类前列腺癌的起源
Differentiation. 2005 Dec;73(9-10):463-73. doi: 10.1111/j.1432-0436.2005.00047.x.
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Cholecalciferol (vitamin D3) inhibits growth and invasion by up-regulating nuclear receptors and 25-hydroxylase (CYP27A1) in human prostate cancer cells.胆钙化醇（维生素D3）通过上调人前列腺癌细胞中的核受体和25-羟化酶（CYP27A1）来抑制生长和侵袭。
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Chemoprevention of prostate cancer by cholecalciferol (vitamin D3): 25-hydroxylase (CYP27A1) in human prostate epithelial cells.胆钙化醇（维生素D3）对前列腺癌的化学预防作用：人前列腺上皮细胞中的25-羟化酶（CYP27A1）
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Modulation of the malignant phenotype of human prostate cancer cells by N-(4-hydroxyphenyl)retinamide (4-HPR).N-(4-羟基苯基)视黄酸（4-HPR）对人前列腺癌细胞恶性表型的调节作用
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Acinar differentiation by non-malignant immortalized human prostatic epithelial cells and its loss by malignant cells.非恶性永生化人前列腺上皮细胞的腺泡分化及其在恶性细胞中的丧失。
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