Italiano G, Magrì V, Paro M, Prosdocimi M
Fidia Research Laboratories, Abano Terme, Italy.
Funct Neurol. 1990 Jul-Sep;5(3):203-5.
This paper briefly describes experimental evidence indicating that bladder dysfunction observed in diabetic rats is due in part to hyperdiuresis and in part to autonomic nerves alterations. The latter, in analogy with somatic nerves alteration, can be ameliorated by treatment of diabetic animals with gangliosides. The use of this agent in the autonomic nerve dysfunction of diabetic origin is discussed.
本文简要描述了实验证据，表明糖尿病大鼠中观察到的膀胱功能障碍部分归因于多尿，部分归因于自主神经改变。与躯体神经改变类似，后者可以通过用神经节苷脂治疗糖尿病动物来改善。本文还讨论了该药物在糖尿病性自主神经功能障碍中的应用。
