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颅内血管瘤破裂致脑内血肿的凝血功能障碍。出血复发的发生率

[Blood coagulation disorders in intracerebral hematoma caused by rupture of intracranial angioma. Incidences on hemorrhagic recurrence].

作者信息

Lamy B, Jourdan C, Leclerc R, Convert J, Deschamps J, Artru F, Tixier S

机构信息

Département d'anesthésie-réanimation, Hôpital neurologique Pierre Wertheimer, Lyon.

出版信息

Agressologie. 1990 Jun;31(6):409-11.

PMID:2285116
Abstract

In 34 patients admitted in Neurological Intensive Care Unit, for a cerebral hematoma by ruptured arterio-venous malformation, are present in 26 patients, on the first day. In many cases, the APTT is increased and this can be respected. These disorders do not seem to modify the incidence of delayed hemorrhage, except for the severe perturbances, related to a DIC or an hepatic deficiency. In these cases, the delayed hemorrhage must be prevented by transfusions of platelets and/or freeze fresh plasma.

摘要

在34例入住神经重症监护病房的患者中,因动静脉畸形破裂导致脑血肿,其中26例在第一天就出现了这种情况。在许多病例中,活化部分凝血活酶时间(APTT)升高,这是可以接受的。除了与弥散性血管内凝血(DIC)或肝脏功能不全相关的严重紊乱外,这些病症似乎并未改变迟发性出血的发生率。在这些情况下,必须通过输注血小板和/或新鲜冰冻血浆来预防迟发性出血。

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