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Interaction of ouabain with (Na+ + K+)ATPase from human heart and from guinea-pig heart.

作者信息

De Pover A, Godfraind T

出版信息

Biochem Pharmacol. 1979 Oct 15;28(20):3051-6. doi: 10.1016/0006-2952(79)90612-9.

DOI:10.1016/0006-2952(79)90612-9
PMID:229866
Abstract
摘要

相似文献

1
Interaction of ouabain with (Na+ + K+)ATPase from human heart and from guinea-pig heart.哇巴因与人心脏及豚鼠心脏的（钠+钾）ATP酶的相互作用。
Biochem Pharmacol. 1979 Oct 15;28(20):3051-6. doi: 10.1016/0006-2952(79)90612-9.
2
Age-associated differences in guinea pig myocardial Na+, K+-ATPase activity and ouabain inhibition and in Mg2+-ATPase activity.豚鼠心肌中钠钾ATP酶活性、哇巴因抑制作用及镁ATP酶活性的年龄相关性差异。
Pharmacology. 1980;21(3):193-7. doi: 10.1159/000137432.
3
Studies on (Na+ plus K+)-activated ATPase. XXXVII. Stabilization by cations of the enzyme-ouabain complex formed with Mg1+ and inorganic phosphate.（钠钾）激活的ATP酶的研究。三十七。阳离子对与Mg1 +和无机磷酸盐形成的酶 - 哇巴因复合物的稳定作用。
Biochim Biophys Acta. 1976 Jan 8;419(1):137-49. doi: 10.1016/0005-2736(76)90378-3.
4
Decreases in active sodium pumping sites and their interaction with ouabain caused by low Na+ incubation of isolated guinea-pig atrial muscle.豚鼠离体心房肌在低钠孵育条件下活性钠泵位点的减少及其与哇巴因的相互作用。
J Pharmacol Exp Ther. 1983 Jun;225(3):660-6.
5
Tissue concentration of Na+, K+-adenosine triphosphatase and the positive inotropic action of ouabain in guinea-pig heart.豚鼠心脏中钠钾-三磷酸腺苷酶的组织浓度及哇巴因的正性肌力作用
J Pharmacol Exp Ther. 1981 Jun;217(3):701-7.
6
Enhancement of cardiac actions of ouabain and its binding to Na+, K+-adenosine triphosphatase by increased sodium influx in isolated guinea-pig heart.通过增加离体豚鼠心脏的钠内流增强哇巴因的心脏作用及其与钠钾三磷酸腺苷酶的结合
J Pharmacol Exp Ther. 1982 Nov;223(2):490-6.
7
Amiodarone: biochemical evidence for its interaction with myocardial Na(+)-K(+)-ATPase in guinea pig microsomal preparations.胺碘酮：豚鼠微粒体制剂中其与心肌钠钾ATP酶相互作用的生化证据。
Biochem Pharmacol. 1991 Feb 1;41(3):470-2. doi: 10.1016/0006-2952(91)90550-o.
8
Identification with potassium and vanadate of two classes of specific ouabain binding sites in a (Na+ + K+) ATPase preparation from the guinea-pig heart.用钾和钒酸盐鉴定豚鼠心脏(Na + + K+)ATP酶制剂中两类特异性哇巴因结合位点。
Biochem Pharmacol. 1980 Apr 15;29(8):1195-9. doi: 10.1016/0006-2952(80)90418-9.
9
The use of a non-linear regression approach for the analysis of the ouabain-K+ interaction with (Na+ + K+)-ATPase from guinea pig and rat hearts.采用非线性回归方法分析哇巴因与豚鼠和大鼠心脏（钠 + 钾）-ATP酶的钾离子相互作用。
Braz J Med Biol Res. 1989;22(4):433-45.
10
Digitalis structure-activity relationship analyses. Conclusions from indirect binding studies with cardiac (Na+ + K+)-ATPase.洋地黄结构-活性关系分析。与心脏（钠+钾）-ATP酶间接结合研究的结论。
Biochem Pharmacol. 1983 Sep 15;32(18):2767-74. doi: 10.1016/0006-2952(83)90090-4.

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Divergent Electrophysiologic Effects of Sacubitril in Digitalis- and Pinacidil-Related Shortened Repolarization: Experimental Evidence for Harmful Effects of Digitalis Glycosides.沙库巴曲对洋地黄和匹那地尔相关的复极缩短的不同电生理效应：洋地黄苷有害作用的实验证据
Pharmaceutics. 2025 Mar 6;17(3):338. doi: 10.3390/pharmaceutics17030338.
2
The Influence of Na(+), K(+)-ATPase on Glutamate Signaling in Neurodegenerative Diseases and Senescence.钠钾ATP酶对神经退行性疾病和衰老过程中谷氨酸信号传导的影响
Front Physiol. 2016 Jun 2;7:195. doi: 10.3389/fphys.2016.00195. eCollection 2016.
3
Effect of prolactin on 86Rb+ uptake, potassium content and [G-3H]ouabain binding of lactating rabbit mammary tissue.
催乳素对泌乳兔乳腺组织86Rb+摄取、钾含量及[G-3H]哇巴因结合的影响。
J Physiol. 1983 Jan;334:1-17. doi: 10.1113/jphysiol.1983.sp014476.
4
Influence of 16 beta formylation on Na, K-ATPase inhibition by cardiac glycosides.16β-甲酰化对强心苷抑制钠钾ATP酶的影响。
Naunyn Schmiedebergs Arch Pharmacol. 1982 Nov;321(2):135-9. doi: 10.1007/BF00518481.
5
The electrogenic sodium pump in guinea-pig ventricular muscle: inhibition of pump current by cardiac glycosides.豚鼠心室肌中的生电钠泵：强心苷对泵电流的抑制作用。
J Physiol. 1982 Sep;330:243-64. doi: 10.1113/jphysiol.1982.sp014339.
6
Renal hypertensive hypertrophy in the rat: a substrate for arrhythmogenicity.大鼠肾性高血压肥大：心律失常的一个基质。
Basic Res Cardiol. 1986 Jan-Feb;81(1):10-9. doi: 10.1007/BF01907423.
7
Role of Na-H exchange in the inotropic action of Bay K 8644 and of ouabain in guinea-pig isolated atria.钠-氢交换在豚鼠离体心房中Bay K 8644和哇巴因正性肌力作用中的作用
Br J Pharmacol. 1990 Aug;100(4):717-22. doi: 10.1111/j.1476-5381.1990.tb14081.x.