National University of Singapore, Singapore.
Biol Psychol. 2013 Feb;92(2):205-11. doi: 10.1016/j.biopsycho.2012.08.010. Epub 2012 Oct 9.
We examined the consistency of results obtained when examining the relationship between self-enhancement and cardiovascular reactivity via analysing raw cardiovascular data and compared this with the hemodynamic profile-compensation deficit (HP-CD) model of blood pressure regulation (James et al., 2012) method. A sample of 112 male participants underwent a computer-based task three times in three weeks. Our results indicate that significant results for self-enhancement and hemodynamic cardiovascular reactivity found using raw cardiovascular data were non-significant when HP-CD model was used. Furthermore, the HP-CD model also obfuscates significant baseline changes with reactivity. We also found that the correlations between blood pressure reactivity and hemodynamic processes obtained in the laboratory setting was reduced rather than enhanced with the use of the HP-CD model. Our results suggest that the HP-CD model should be used cautiously and could contribute to inconsistent results when examining the role of psychological factors in biological outcomes.
我们通过分析原始心血管数据来检查自我增强与心血管反应性之间关系时所获得的结果的一致性,并将其与血压调节的血液动力学特征补偿不足 (HP-CD) 模型 (James 等人,2012) 方法进行了比较。112 名男性参与者的样本在三周内进行了三次基于计算机的任务。我们的结果表明,使用原始心血管数据时,自我增强和血液动力学心血管反应性的显著结果在使用 HP-CD 模型时变得不显著。此外,HP-CD 模型还混淆了与反应性相关的显著基线变化。我们还发现,使用 HP-CD 模型后,实验室环境中获得的血压反应性与血液动力学过程之间的相关性降低,而不是增强。我们的结果表明,在检查心理因素对生物学结果的作用时,应谨慎使用 HP-CD 模型,并且可能会导致结果不一致。