Bol'shakov V Iu, Sverdlov A G
Radiobiologiia. 1990 Jan-Feb;30(1):94-7.
Irradiation of isolated frog sympathetic ganglia IX-X with high doses caused a long-term depolarization of a single ganglion cell and increase of synaptic activation threshold. The radiation effects observed were not accompanied by the transmission failure. The authors suggest that the changes in the synaptic mechanisms play a minor role in the development of the CNS syndrome.
用高剂量照射离体青蛙交感神经节IX - X,可导致单个神经节细胞长期去极化,并增加突触激活阈值。所观察到的辐射效应并未伴随传递失败。作者认为,突触机制的变化在中枢神经系统综合征的发展中起次要作用。