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降低茉莉酸水平会导致 ein2 突变体对乙烯产生反应。

Reducing jasmonic acid levels causes ein2 mutants to become ethylene responsive.

机构信息

Department of Biochemistry, Cellular and Molecular Biology, University of Tennessee, Knoxville, TN 37917, USA.

出版信息

FEBS Lett. 2013 Jan 16;587(2):226-30. doi: 10.1016/j.febslet.2012.11.030. Epub 2012 Dec 7.

DOI:10.1016/j.febslet.2012.11.030
PMID:23219920
Abstract

It has previously been shown that jasmonic acid affects the ethylene signaling pathway. EIN2 is a central component of ethylene signaling that is downstream of the receptors. EIN2 has previously been shown to be required for ethylene responses. We found that reducing jasmonic acid levels, either mutationally or chemically, caused ein2 ethylene-insensitive mutants to become ethylene responsive. This effect was not seen with the ethylene-insensitive etr1-1 mutants that affect receptor function. Based upon these results, we propose a model where jasmonic acid is inhibiting ethylene signal transduction down-stream of the ethylene receptors. This may involve an EIN2-independent pathway.

摘要

先前已经表明,茉莉酸会影响乙烯信号通路。EIN2 是乙烯信号转导的核心组成部分,位于受体的下游。先前的研究表明,EIN2 对于乙烯反应是必需的。我们发现,无论是突变还是化学方法降低茉莉酸水平,都会导致 ein2 乙烯不敏感突变体对乙烯产生反应。而对于影响受体功能的乙烯不敏感 etr1-1 突变体,则不会出现这种情况。基于这些结果,我们提出了一个模型,其中茉莉酸抑制乙烯受体下游的乙烯信号转导。这可能涉及到一条不依赖于 EIN2 的途径。

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