Price J
Clin Chim Acta. 1978 Feb 1;83(1-2):61-5. doi: 10.1016/0009-8981(78)90207-3.
It is demonstrated that the increased tryptaminuria which follows alcohol ingestion can be abolished by the concurrent ingestion of alkali even when the amount of alkali consumed is insufficient to increase urinary pH to 6.5 (above which pH a fall in urinary tryptamine may be anticipated, from previous studies, to occur). It is suggested that this increased tryptaminuria is largely if not wholly dependent on the metabolic acidosis induced by alcohol which the concurrent ingestion of alkali abolishes.
结果表明，饮酒后出现的色胺尿增加现象，即使同时摄入的碱量不足以使尿液pH值升高到6.5（根据以往研究，超过此pH值预计尿液中色胺会减少），也可通过同时摄入碱而消除。有人提出，这种色胺尿增加现象即使不是完全依赖于酒精诱导的代谢性酸中毒，也是在很大程度上依赖于此，而同时摄入碱可消除这种代谢性酸中毒。
