Comment on 'cytotoxic effect of disulfiram/copper on human glioblastoma cell lines and ALDH-positive cancer-stem-like cells'.
作者信息
Cvek B
出版信息
Br J Cancer. 2013 Mar 5;108(4):993. doi: 10.1038/bjc.2013.18. Epub 2013 Jan 22.
Abstract
摘要
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Comment on 'cytotoxic effect of disulfiram/copper on human glioblastoma cell lines and ALDH-positive cancer-stem-like cells'.关于“双硫仑/铜对人胶质母细胞瘤细胞系及醛脱氢酶阳性癌干细胞样细胞的细胞毒性作用”的评论
Br J Cancer. 2013 Mar 5;108(4):993. doi: 10.1038/bjc.2013.18. Epub 2013 Jan 22.
2
Reply: Cytotoxic effect of disulfiram/copper on human glioblastoma cell lines and ALDH-positive cancer-stem-like cells.回复：双硫仑/铜对人胶质母细胞瘤细胞系和醛脱氢酶阳性癌干细胞样细胞的细胞毒性作用。
Br J Cancer. 2013 Mar 5;108(4):994. doi: 10.1038/bjc.2013.19. Epub 2013 Jan 22.
3
Cytotoxic effect of disulfiram/copper on human glioblastoma cell lines and ALDH-positive cancer-stem-like cells.双硫仑/铜对人神经胶质瘤细胞系和 ALDH 阳性癌干细胞样细胞的细胞毒性作用。
Br J Cancer. 2012 Oct 23;107(9):1488-97. doi: 10.1038/bjc.2012.442. Epub 2012 Oct 2.
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Inhibitory effect on ovarian cancer ALDH+ stem-like cells by Disulfiram and Copper treatment through ALDH and ROS modulation.通过调节 ALDH 和 ROS，二硫化四乙基秋兰姆和铜处理对卵巢癌 ALDH+ 肿瘤干细胞的抑制作用。
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Disulfiram/copper targets stem cell-like ALDH population of multiple myeloma by inhibition of ALDH1A1 and Hedgehog pathway.双硫仑/铜通过抑制醛脱氢酶1A1（ALDH1A1）和刺猬信号通路来靶向多发性骨髓瘤的干细胞样醛脱氢酶群体。
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Suppressing glioblastoma stem cell function by aldehyde dehydrogenase inhibition with chloramphenicol or disulfiram as a new treatment adjunct: an hypothesis.用氯霉素或双硫仑抑制醛脱氢酶来抑制神经胶质瘤干细胞功能作为一种新的治疗辅助手段：一种假说。
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A2780 human ovarian cancer cells with acquired paclitaxel resistance display cancer stem cell properties.具有获得性紫杉醇耐药性的A2780人卵巢癌细胞表现出癌症干细胞特性。
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Reply: Cytotoxic effect of disulfiram/copper on human glioblastoma cell lines and ALDH-positive cancer-stem-like cells.回复：双硫仑/铜对人胶质母细胞瘤细胞系和醛脱氢酶阳性癌干细胞样细胞的细胞毒性作用。
Br J Cancer. 2013 Mar 5;108(4):994. doi: 10.1038/bjc.2013.19. Epub 2013 Jan 22.
本文引用的文献
1
Cytotoxic effect of disulfiram/copper on human glioblastoma cell lines and ALDH-positive cancer-stem-like cells.双硫仑/铜对人神经胶质瘤细胞系和 ALDH 阳性癌干细胞样细胞的细胞毒性作用。
Br J Cancer. 2012 Oct 23;107(9):1488-97. doi: 10.1038/bjc.2012.442. Epub 2012 Oct 2.
2
Nonprofit drugs as the salvation of the world's healthcare systems: the case of Antabuse (disulfiram).非营利性药物作为世界医疗体系的救星：以双硫仑（戒酒硫）为例。
Drug Discov Today. 2012 May;17(9-10):409-12. doi: 10.1016/j.drudis.2011.12.010. Epub 2011 Dec 16.
3
Targeting malignancies with disulfiram (Antabuse): multidrug resistance, angiogenesis, and proteasome.用双硫仑（安塔布司）靶向恶性肿瘤：多药耐药性、血管生成和蛋白酶体。
Curr Cancer Drug Targets. 2011 Mar;11(3):332-7. doi: 10.2174/156800911794519806.
4
The status of disulfiram: a half of a century later.双硫仑的现状：半个世纪之后
J Clin Psychopharmacol. 2006 Jun;26(3):290-302. doi: 10.1097/01.jcp.0000222512.25649.08.
5
Sodium dithiocarb as adjuvant immunotherapy for high risk breast cancer: a randomized study.二硫代氨基甲酸钠作为高危乳腺癌辅助免疫疗法的随机研究。
Biotherapy. 1993;6(1):9-12. doi: 10.1007/BF01877380.
6
A review of the pharmacokinetics and pharmacodynamics of disulfiram and its metabolites.双硫仑及其代谢产物的药代动力学和药效学综述。
Acta Psychiatr Scand Suppl. 1992;369:15-26. doi: 10.1111/j.1600-0447.1992.tb03310.x.
7
Spontaneous regression of breast cancer.
Prog Clin Biol Res. 1977;12:47-53.
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