Amphetamine mechanisms and actions at the dopamine terminal revisited.
作者信息
Calipari Erin S, Ferris Mark J
机构信息
Neuroscience Program and Department of Physiology and Pharmacology, Wake Forest School of Medicine, Winston-Salem, North Carolina 27157, USA.
出版信息
J Neurosci. 2013 May 22;33(21):8923-5. doi: 10.1523/JNEUROSCI.1033-13.2013.
Abstract
摘要
相似文献
1
Amphetamine mechanisms and actions at the dopamine terminal revisited.重新审视安非他命在多巴胺终端的作用机制与行动。
J Neurosci. 2013 May 22;33(21):8923-5. doi: 10.1523/JNEUROSCI.1033-13.2013.
2
Endogenous opioids in dopaminergic cell body regions modulate amphetamine-induced increases in extracellular dopamine levels in the terminal regions.多巴胺能细胞体区域的内源性阿片类物质调节苯丙胺引起的终末区域细胞外多巴胺水平的升高。
J Pharmacol Exp Ther. 2002 Mar;300(3):932-8. doi: 10.1124/jpet.300.3.932.
3
Dopamine uptake inhibitors but not dopamine releasers induce greater increases in motor behavior and extracellular dopamine in adolescent rats than in adult male rats.多巴胺摄取抑制剂而非多巴胺释放剂在青少年大鼠中引起的运动行为和细胞外多巴胺增加幅度大于成年雄性大鼠。
J Pharmacol Exp Ther. 2010 Oct;335(1):124-32. doi: 10.1124/jpet.110.167320. Epub 2010 Jul 6.
4
High doses of amphetamine augment, rather than disrupt, exocytotic dopamine release in the dorsal and ventral striatum of the anesthetized rat.高剂量安非他命增强而不是破坏麻醉大鼠背侧和腹侧纹状体的胞吐多巴胺释放。
J Neurochem. 2011 Dec;119(6):1162-72. doi: 10.1111/j.1471-4159.2011.07407.x. Epub 2011 Aug 25.
5
The roles of dopamine transport inhibition and dopamine release facilitation in wake enhancement and rebound hypersomnolence induced by dopaminergic agents.多巴胺转运体抑制和多巴胺释放促进在多巴胺能药物诱导的觉醒增强和反弹性过度嗜睡中的作用。
Sleep. 2009 Nov;32(11):1425-38. doi: 10.1093/sleep/32.11.1425.
6
Amphetamine distorts stimulation-dependent dopamine overflow: effects on D2 autoreceptors, transporters, and synaptic vesicle stores.苯丙胺会扭曲依赖刺激的多巴胺释放：对D2自身受体、转运体和突触囊泡储存的影响。
J Neurosci. 2001 Aug 15;21(16):5916-24. doi: 10.1523/JNEUROSCI.21-16-05916.2001.
7
Amphetamine sensitization of stress-induced turning in animals given unilateral dopamine transplants in infancy.
Brain Res. 1990 Apr 23;514(1):143-6. doi: 10.1016/0006-8993(90)90447-j.
8
Fluctuation of the dopamine uptake inhibition potency of cocaine, but not amphetamine, at mammalian cells expressing the dopamine transporter.可卡因对表达多巴胺转运体的哺乳动物细胞的多巴胺摄取抑制效力存在波动，而苯丙胺则不然。
Brain Res. 2007 Feb 2;1131(1):68-76. doi: 10.1016/j.brainres.2006.11.018. Epub 2006 Dec 13.
9
Time-dependent changes in the sensitivity of dopamine neurons to low doses of apomorphine following amphetamine infusion.
Brain Res. 1989 Mar 27;483(1):17-29. doi: 10.1016/0006-8993(89)90030-9.
10
The effects of blocking catecholamine uptake on amphetamine-induced circling behaviour in mice with unilateral destruction of striatal dopaminergic nerve terminals.
J Pharm Pharmacol. 1976 Jun;28(6):530-2. doi: 10.1111/j.2042-7158.1976.tb02785.x.
引用本文的文献
1
Does Dysregulation Of The Indirect Pathway Contribute To The Pathophysiology Of Catatonia Through Neurotransmitter Imbalance?间接通路的失调是否通过神经递质失衡导致紧张症的病理生理学？
Clin Neuropsychiatry. 2025 Jun;22(3):229-242. doi: 10.36131/cnfioritieditore20250306.
2
Knockout of Bmal1 in dopaminergic neurons induces ADHD-like symptoms via hyperactive dopamine signaling in male mice.敲除雄性小鼠多巴胺能神经元中的Bmal1会通过多巴胺信号过度活跃诱发类似注意力缺陷多动障碍（ADHD）的症状。
Behav Brain Funct. 2025 Jul 11;21(1):22. doi: 10.1186/s12993-025-00287-w.
3
Individuals with methamphetamine use disorder show reduced directed exploration and learning rates independent of an aversive interoceptive state induction.患有甲基苯丙胺使用障碍的个体表现出定向探索和学习率降低，且与厌恶的内感受状态诱导无关。
Commun Psychol. 2025 Jun 7;3(1):90. doi: 10.1038/s44271-025-00269-8.
4
Exploring α-Syn's Functions Through Ablation Models: Physiological and Pathological Implications.通过基因敲除模型探索α-突触核蛋白的功能：生理和病理意义
Cell Mol Neurobiol. 2025 May 19;45(1):44. doi: 10.1007/s10571-025-01560-2.
5
Multifunctionality and Possible Medical Application of the BPC 157 Peptide-Literature and Patent Review.BPC 157肽的多功能性及其可能的医学应用——文献与专利综述
Pharmaceuticals (Basel). 2025 Jan 30;18(2):185. doi: 10.3390/ph18020185.
6
Genome data based deep learning identified new genes predicting pharmacological treatment response of attention deficit hyperactivity disorder.基于基因组数据的深度学习识别出预测注意力缺陷多动障碍药物治疗反应的新基因。
Transl Psychiatry. 2025 Feb 7;15(1):46. doi: 10.1038/s41398-025-03250-5.
7
RLS-associated MEIS transcription factors control distinct processes in human neural stem cells.RLS 相关的 MEIS 转录因子在人类神经干细胞中控制不同的过程。
Sci Rep. 2024 Nov 22;14(1):28986. doi: 10.1038/s41598-024-80266-9.
8
Efficacy and Safety of Modafinil for Treatment of Amphetamine-Type Stimulant Use Disorder: A Systematic Review and Meta-Analysis of Randomized Placebo-Controlled Trials: Efficacité et innocuité du modafinil pour le traitement des troubles liés à l'usage de stimulants de type amphétamine : revue systématique et méta-analyse d'essais randomisés contrôlés par placebo.莫达非尼治疗苯丙胺类兴奋剂使用障碍的疗效和安全性：随机安慰剂对照试验的系统评价和荟萃分析：莫达非尼治疗苯丙胺类兴奋剂使用障碍的疗效和安全性：随机安慰剂对照试验的系统评价和荟萃分析。
Can J Psychiatry. 2024 Nov;69(11):793-805. doi: 10.1177/07067437241262967. Epub 2024 Jul 21.
9
Individuals with Methamphetamine Use Disorder Show Reduced Directed Exploration and Learning Rates Independent of an Aversive Interoceptive State Induction.患有甲基苯丙胺使用障碍的个体表现出定向探索和学习率降低，与厌恶的内感受状态诱导无关。
medRxiv. 2025 Feb 24:2024.05.17.24307491. doi: 10.1101/2024.05.17.24307491.
10
Doxycycline reversal of amphetamine-induced mania-like behavior is related to adjusting brain monoamine abnormalities and antioxidant effects in primary hippocampal neurons.强力霉素逆转安非他命引起的躁狂样行为与调节原代海马神经元脑单胺异常和抗氧化作用有关。
Naunyn Schmiedebergs Arch Pharmacol. 2024 Aug;397(8):6017-6035. doi: 10.1007/s00210-024-03009-7. Epub 2024 Feb 22.
本文引用的文献
1
Amphetamine paradoxically augments exocytotic dopamine release and phasic dopamine signals.安非他命反常地增强了多巴胺的胞吐释放和相位性多巴胺信号。
J Neurosci. 2013 Jan 9;33(2):452-63. doi: 10.1523/JNEUROSCI.2136-12.2013.
2
Cocaine self-administration produces pharmacodynamic tolerance: differential effects on the potency of dopamine transporter blockers, releasers, and methylphenidate.可卡因自我给药会产生药效学耐受性：对多巴胺转运体阻滞剂、释放剂和哌醋甲酯效力的差异影响。
Neuropsychopharmacology. 2012 Jun;37(7):1708-16. doi: 10.1038/npp.2012.17. Epub 2012 Mar 7.
3
New insights into the mechanism of action of amphetamines.对安非他明作用机制的新见解。
Annu Rev Pharmacol Toxicol. 2007;47:681-98. doi: 10.1146/annurev.pharmtox.47.120505.105140.
4
Inhibition of dopamine efflux in the rat nucleus accumbens during abstinence after free access to d-amphetamine.在大鼠自由获取右旋苯丙胺后戒断期间，伏隔核中多巴胺外流的抑制作用。
Behav Brain Res. 2002 Jan 7;128(1):1-12. doi: 10.1016/s0166-4328(01)00265-0.
5
Amphetamine distorts stimulation-dependent dopamine overflow: effects on D2 autoreceptors, transporters, and synaptic vesicle stores.苯丙胺会扭曲依赖刺激的多巴胺释放：对D2自身受体、转运体和突触囊泡储存的影响。
J Neurosci. 2001 Aug 15;21(16):5916-24. doi: 10.1523/JNEUROSCI.21-16-05916.2001.
6
Mechanisms of amphetamine action revealed in mice lacking the dopamine transporter.在缺乏多巴胺转运体的小鼠中揭示的苯丙胺作用机制。
J Neurosci. 1998 Mar 15;18(6):1979-86. doi: 10.1523/JNEUROSCI.18-06-01979.1998.
7
Anatomical and affinity state comparisons between dopamine D1 and D2 receptors in the rat central nervous system.大鼠中枢神经系统中多巴胺D1和D2受体之间的解剖学及亲和力状态比较。
Neuroscience. 1989;30(3):767-77. doi: 10.1016/0306-4522(89)90168-1.
8
Weak base model of amphetamine action.
Ann N Y Acad Sci. 1992 Jun 28;654:525-8. doi: 10.1111/j.1749-6632.1992.tb26020.x.
9
Kinetics and metabolism of amphetamine in the brain of rats of different ages.不同年龄大鼠脑中苯丙胺的动力学和代谢
Naunyn Schmiedebergs Arch Pharmacol. 1975;291(2):111-21. doi: 10.1007/BF00500043.
Zotero 插件